Claim · model-written summary
CaTFT4 silencing enhanced growth of avirulent Xcv in pepper.
- Taxon
- peppere0
- Treatment
- silencing of CaTFT4e0
- Interactor
- Xcve0pathogen
- Gene
- CaTFT4e0
- Outcome
- growth of virulent and avirulent Xcve0
- Direction
- enhancede0
- Qualifier
- avirulente0
Not reportedMaterialCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValue
Verbatim
e0“silencing of CaTFT4 in pepper delayed the appearance of ETI-associated cell death and enhanced growth of virulent and avirulent Xcv”
Title
Doron Teper; Dor Salomon; Sukumaran Sunitha; et al. The Plant Journal 2013. doi:10.1111/tpj.12391
Other claims from this paper (13)
- Mechanism XopQ suppresses ETI-associated cell death induced by MAP kinase cascade components and R/avr pairs.
- Mechanism XopQ inhibits ETI-associated cell death in resistant pepper challenged with avirulent Xcv.
- Observation XopQ enhances bacterial growth in resistant pepper.
- Observation XopQ enhances bacterial growth in resistant tomato.
- Observation Tomato SlTFT4 and homologs interact with XopQ.
- Intervention effect Mutation of the XopQ binding site impaired interaction with CaTFT4 in yeast.
- Intervention effect Mutation of the XopQ binding site impaired virulence in planta.
- Observation TFT4 mRNA abundance increased during incompatible tomato interaction with Xcv.
- Observation TFT4 mRNA abundance increased during incompatible pepper interaction with Xcv.
- Intervention effect Silencing NbTFT4 reduced MAPKKKα-induced cell death in Nicotiana benthamiana.
- Intervention effect CaTFT4 silencing delayed ETI-associated cell death in pepper.
- Intervention effect CaTFT4 silencing enhanced growth of virulent Xcv in pepper.
- Mechanism The authors suggest XopQ suppresses ETI and immunity-associated cell death through TFT4 interaction.