Claim · model-written summary
Expression of canonical α-SNAPs counteracted cytotoxicity caused by resistance-type α-SNAPs.
- Taxon
- Soybean
Glycine maxt0 - Treatment
- Expression of these α-SNAPse0
- Gene
- α-SNAPse0
- Outcome
- cytotoxicitye0
- Direction
- counteractede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Verbatim
e0“Expression of these α-SNAPs counteracted the cytotoxicity of resistance-type Rhg1 α-SNAPs”
t0“soybean”
Title
Adam M. Bayless; John M. Smith; Junqi Song; et al. Proceedings of the National Academy of Sciences 2016. doi:10.1073/pnas.1610150113
Other claims from this paper (7)
- Observation Resistance-type Rhg1 α-SNAP interacted defectively with NSF.
- Intervention effect Elevated resistance-type Rhg1 α-SNAP expression depleted SNARE-recycling 20S complexes.
- Intervention effect Elevated resistance-type Rhg1 α-SNAP expression disrupted vesicle trafficking.
- Intervention effect Elevated resistance-type Rhg1 α-SNAP expression increased NSF abundance.
- Intervention effect Elevated resistance-type Rhg1 α-SNAP expression caused cytotoxicity.
- Observation Resistance-type α-SNAPs hyperaccumulated at nematode feeding sites relative to wild-type α-SNAPs.
- Mechanism Hyperaccumulation of resistance-type α-SNAPs promoted demise of the feeding interface.