Claim · model-written summary
T3 induced drought tolerance through stress-responsive gene expression and biosynthesis pathways.
- Taxon
- tobacco
Nicotiana tabacume1 - Treatment
- T3 drought-hardening treatmente0
- Outcome
- drought tolerancee0
- Qualifier
- expression of various stress-responsive genese0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“T3 drought-hardening treatment-induced drought tolerance via the expression of various stress-responsive genes by triggering the biosynthesis pathways of proline (P5CS1), polyamines (ADC2), ABA-dependent (SnRK2, AREB1), and independent pathways (DREB2B), and antioxidant defense-related genes (CAT, APX1, GR2) in response to drought stress.”
e1“two tobacco varieties namely HongHuaDaJinYuan (H) and Yun Yan-100 (Y)”
Title
Rayyan Khan; Xinghua Ma; Shahen Shah; et al. BMC Plant Biology 2020. doi:10.1186/s12870-020-02688-7
Other claims from this paper (8)
- Intervention effect T2 hardening significantly reduced growth under drought stress.
- Intervention effect T3 hardening significantly reduced growth under drought stress.
- Intervention effect T3 improved POD, CAT and APX enzymatic defenses.
- Intervention effect T3 improved AsA nonenzymatic defense.
- Intervention effect T3 elevated proline levels.
- Intervention effect T3 elevated soluble sugar levels.
- Intervention effect Hardening treatments differentially regulated genes in both tobacco varieties.
- Intervention effect Drought-hardening improved drought tolerance and adaptation in the two tobacco varieties.