Claim · model-written summary
The SA response of Pathogenesis-Related genes was compromised under low CO2.
- Taxon
- Arabidopsis
Arabidopsise2 - Gene
- Pathogenesis-Related genese0
- Outcome
- SA response of Pathogenesis-Related genese0
- Direction
- compromisede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“the SA response of Pathogenesis-Related genes was compromised.”
e1“under low CO2 conditions, although the SA response of Pathogenesis-Related genes was compromised.”
e2“Arabidopsis plants”
Title
Role of salicylic acid in low CO2 response in Arabidopsis
Kosei Yoneda; Susumu Uehara; Takakazu Matsuura; et al. Plant and Cell Physiology 2025. doi:10.1093/pcp/pcaf052
Other claims from this paper (12)
- Intervention effect Low CO2 induced salicylic acid in Arabidopsis.
- Intervention effect Low CO2 induced abscisic acid in Arabidopsis.
- Intervention effect Low CO2 dramatically increased SA.
- Observation SA induction was abolished in the SA induction-deficient2 mutant.
- Mechanism The SA mutant result indicates SA is primarily synthesized by isochorismate synthase under low CO2.
- Intervention effect ABA level increased under low CO2.
- Observation Low CO2 altered expression of genes involved in SA and ABA accumulation.
- Intervention effect Low CO2 increased expression of SA- and ABA-responsive genes.
- Observation Low CO2 altered programmed cell death-associated gene expression.
- Intervention effect Low CO2 suppressed photosynthesis-associated gene expression.
- Observation High SA accumulation further manifested the gene responses.
- Association SA increase appears associated with suppression of nonessential genes and altered cell-death genes.