Claim · model-written summary
High Mo levels modulated hormone signaling in foxtail millet.
- Taxon
- foxtail millet
Setaria italicae6 - Treatment
- High Mo levelse5
- Outcome
- hormone signalinge6
- Qualifier
- modulatede6
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“Setaria Italica L.”
e1“low concentrations of Mo increased the growth of foxtail millet”
e5“High Mo levels up-regulated key carotenoid biosynthesis genes (such as 9-cis-epoxycarotenoid dioxygenase 4, NCED4, 9-cis-epoxycarotenoid dioxygenase 5, NCED5, zeta-carotene desaturase, ZDS)”
e6“modulated hormone signaling, optimizing starch-sucrose regulation and boosting stress resilience in foxtail millet”
Title
Meijun Guo; Mengmeng Sun; Yaqing Bai; et al. Plant Growth Regulation 2026. doi:10.1007/s10725-025-01406-3
Other claims from this paper (9)
- Intervention effect Low Mo concentrations increased the growth of foxtail millet.
- Intervention effect High Mo concentrations suppressed the growth of foxtail millet.
- Intervention effect The 8 mg L− 1 Mo treatment produced more differentially expressed genes than other concentrations.
- Intervention effect The 15 mg L− 1 Mo treatment inhibited starch and sucrose metabolism compared with 8 mg L− 1.
- Intervention effect The 15 mg L− 1 Mo treatment enhanced phenylpropanoid and flavonoid biosynthesis compared with 8 mg L− 1.
- Intervention effect High Mo levels up-regulated key carotenoid biosynthesis genes.
- Mechanism High Mo levels boosted stress resilience in foxtail millet.
- Observation RNA-seq of foxtail millet under varying Mo levels identified crucial pathways including phenylpropanoid synthesis, starch metabolism and hormone signaling.
- Mechanism Supra-optimal Mo affected foxtail millet physiology through carbon and nitrogen cycle imbalance and antioxidant impairment, ultimately suppressing growth.