Claim · model-written summary
Higher light intensities switch cytokinin from inhibiting to promoting hypocotyl elongation.
- Taxon
- Arabidopsis
Arabidopsise1 - Treatment
- higher light intensitiese0
- Outcome
- hypocotyl elongatione0
- Direction
- switche0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“higher light intensities causing a switch in the response to cytokinin from an inhibitor to a promoter of hypocotyl elongation.”
e1“dark-grown Arabidopsis plants”
Title
Anne Cortleven; Stephanie Ehret; Thomas Schmülling; et al. Journal of Experimental Botany 2018. doi:10.1093/jxb/ery344
Other claims from this paper (5)
- Mechanism Cytokinin inhibition of hypocotyl elongation is largely independent of ethylene.
- Mechanism The cytokinin signal is mainly mediated by ARABIDOPSIS HISTIDINE KINASE3.
- Mechanism The cytokinin signal is mediated by ARABIDOPSIS RESPONSE REGULATOR1 together with ARR12.
- Intervention effect Mutation of COP1, DE-ETIOLATED1, or CYTOKININ INSENSITIVE4/COP10 makes plants insensitive to cytokinin.
- Mechanism The light-signaling factors are indispensable for transcriptional response during cytokinin-induced de-etiolation.