Claim · model-written summary
Recovery may be largely independent of the inhibitor and auxins and due to another growth factor limiting cell length.
- Taxon
- Pisum sativum
Lathyrus oleraceuse0 - Outcome
- Recoverye7
- Qualifier
- another growth factor limiting cell lengthe7
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“roots of Pisum sativum seedlings”
e7“Recovery may be very largely independent of both inhibitor and auxins and due to the action of another growth factor limiting cell length”
Title
L. J. AUDUS; M. E. BROWNBRIDGE Journal of Experimental Botany 1957. doi:10.1093/jxb/8.1.105
Other claims from this paper (13)
- Observation Positive curvature started at a maximum rate of 0.30–0.32 deg./min.
- Observation Positive curvature had a reaction time of 11.5 minutes.
- Observation Recovery was complete after a further 150–200 minutes at a similar rate of curvature.
- Observation Overall root growth-rates were reduced during the phase of positive curvature.
- Intervention effect Low concentrations of both auxins increased the rate of positive curvature by 30–40 per cent.
- Intervention effect Low concentrations of both auxins shortened the reaction time roughly in proportion.
- Intervention effect Low concentrations of both auxins increased growth-rates of both sides of the root to the same extent.
- Intervention effect High concentrations of both auxins reduced the rates of curvature by 50 per cent.
- Intervention effect High concentrations of both auxins lengthened the reaction time.
- Intervention effect High concentrations of both auxins inhibited the growth of both sides of the root.
- Intervention effect Neither auxin concentration otherwise affected the time course of response and recovery.
- Mechanism Geotropic response is suggested to be due to de novo production of an endogenous inhibitor.
- Mechanism The inhibitor is suggested not to be indole-3-acetic acid or any similar compound.