Claim · model-written summary
Treatment with buthionine sulfoximine (a GSH inhibitor) reduced the expression of ROS-regulating genes in N. benthamiana.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianae0 - Treatment
- treatment with buthionine sulfoximine (a GSH inhibitor)e4
- Gene
- ROS-regulating genese4
- Outcome
- expression of ROS-regulating genese4
- Direction
- reducede4
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“plant viral resistance in Nicotiana benthamiana”
e4“However, treatment with buthionine sulfoximine (a GSH inhibitor) accelerated oxidative damage, elevated ROS accumulation, compromised plant systemic resistance, suppressed the SA-mediated signalling pathway, and reduced the expression of ROS-regulating genes”
Title
Feng Zhu; Qi‐Ping Zhang; Yan‐Ping Che; et al. Molecular Plant Pathology 2021. doi:10.1111/mpp.13138
Other claims from this paper (19)
- Intervention effect TMV infection increased the expression of GSH biosynthesis genes NbECS and NbGS and GSH content in N. benthamiana.
- Intervention effect Silencing of NbECS or NbGS accelerated oxidative damage in N. benthamiana.
- Intervention effect Silencing of NbECS or NbGS increased ROS accumulation in N. benthamiana.
- Intervention effect Silencing of NbECS or NbGS compromised plant resistance to TMV.
- Intervention effect Silencing of NbECS or NbGS suppressed the SA-mediated signalling pathway.
- Intervention effect Application of GSH or its activator alleviated oxidative damage in N. benthamiana.
- Intervention effect Application of GSH or its activator decreased ROS accumulation in N. benthamiana.
- Intervention effect Application of GSH or its activator elevated local and systemic resistance of N. benthamiana.
- Intervention effect Application of GSH or its activator enhanced the SA-mediated signalling pathway in N. benthamiana.
- Intervention effect Application of GSH or its activator increased the expression of ROS scavenging-related genes in N. benthamiana.
- Intervention effect Treatment with buthionine sulfoximine (a GSH inhibitor) accelerated oxidative damage in N. benthamiana.
- Intervention effect Treatment with buthionine sulfoximine (a GSH inhibitor) elevated ROS accumulation in N. benthamiana.
- Intervention effect Treatment with buthionine sulfoximine (a GSH inhibitor) compromised systemic resistance of N. benthamiana.
- Intervention effect Treatment with buthionine sulfoximine (a GSH inhibitor) suppressed the SA-mediated signalling pathway in N. benthamiana.
- Intervention effect Overexpression of NbECS reduced oxidative damage in N. benthamiana.
- Intervention effect Overexpression of NbECS decreased ROS accumulation in N. benthamiana.
- Intervention effect Overexpression of NbECS increased resistance of N. benthamiana to TMV.
- Intervention effect Overexpression of NbECS activated the SA-mediated signalling pathway in N. benthamiana.
- Intervention effect Overexpression of NbECS increased the expression of ROS scavenging-related genes in N. benthamiana.