Claim · model-written summary
GH3.5 overexpression raised SA accumulation after avirulent pathogen challenge.
- Taxon
- Arabidopsis thaliana
Arabidopsis thalianae1 - Material
- gh3.5-1De0
- Treatment
- Overexpression of the GH3.5 genee0
- Gene
- GH3.5e0
- Outcome
- accumulation of SAe0
- Direction
- elevatede0
Not reportedCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Overexpression of the GH3.5 gene in an activation-tagged mutant gh3.5-1D led to elevated accumulation of SA and increased expression of PR-1 in local and systemic tissues in response to avirulent pathogens”
e1“Arabidopsis (Arabidopsis thaliana)”
Title
Zhongqin Zhang; Qun Li; Zhimiao Li; et al. PLANT PHYSIOLOGY 2007. doi:10.1104/pp.107.106021
Other claims from this paper (10)
- Observation GH3.5 protein showed in vitro adenylation activity on IAA and SA.
- Intervention effect GH3.5 overexpression increased PR-1 expression locally and systemically after avirulent pathogen challenge.
- Intervention effect GH3.5 insertion mutations partially compromised systemic acquired resistance.
- Intervention effect GH3.5 insertion mutations diminished systemic PR-1 expression.
- Observation The gh3.5-1D mutant accumulated high free IAA after pathogen infection.
- Observation gh3.5-1D impaired resistance-gene-mediated resistance.
- Observation The GH3.6 activation mutant dfl1-D also impaired resistance-gene-mediated resistance.
- Observation SA and auxin pathways were simultaneously augmented in infected gh3.5-1D.
- Mechanism GH3.5 amplified the SA pathway through induction of SA-responsive genes and basal defense components.
- Mechanism The auxin pathway was derepressed with increased IAA biosynthesis and reduced auxin repressor gene expression.