Claim · model-written summary
ROP3 loss or dominant-negative mutation altered early embryo cell division.
- Taxon
- Arabidopsis
Arabidopsist0 - Treatment
- Loss-of-function and dominant-negative (DN) mutations in ROP3e0
- Gene
- ROP3e0
- Outcome
- cell division patterning during early embryogenesise0
- Direction
- alterede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Loss-of-function and dominant-negative (DN) mutations in ROP3 induced a spectrum of similar defects starting with altered cell division patterning during early embryogenesis”
t0“Arabidopsis”
Title
Jia-bao Huang; Huili Liu; Min Chen; et al. The Plant Cell 2014. doi:10.1105/tpc.114.127902
Other claims from this paper (9)
- Observation ROP3 mutations distorted embryo development and organ formation.
- Intervention effect ROP3 mutations retarded root gravitropism.
- Intervention effect ROP3 mutations reduced auxin-dependent hypocotyl elongation.
- Observation DN-rop3 embryos had reduced expression of AUXIN RESPONSE FACTOR5/MONOPTEROS, PLETHORA1 and PLT2.
- Observation ROP3 mutations altered PIN polar localization at the plasma membrane.
- Observation ROP3 mutations disrupted root auxin maxima.
- Association Auxin induces ROP3.
- Observation ROP3 is prominently detected in root stele cells.
- Mechanism ROP3 maintains PIN polarity, enabling polar auxin transport and plant patterning.