Claim · model-written summary
ROP3 mutations distorted embryo development and organ formation.
- Taxon
- Arabidopsis
Arabidopsist0 - Outcome
- distorted embryo development, defective organ formatione0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“These resulted in distorted embryo development, defective organ formation”
t0“Arabidopsis”
Title
Jia-bao Huang; Huili Liu; Min Chen; et al. The Plant Cell 2014. doi:10.1105/tpc.114.127902
Other claims from this paper (9)
- Intervention effect ROP3 loss or dominant-negative mutation altered early embryo cell division.
- Intervention effect ROP3 mutations retarded root gravitropism.
- Intervention effect ROP3 mutations reduced auxin-dependent hypocotyl elongation.
- Observation DN-rop3 embryos had reduced expression of AUXIN RESPONSE FACTOR5/MONOPTEROS, PLETHORA1 and PLT2.
- Observation ROP3 mutations altered PIN polar localization at the plasma membrane.
- Observation ROP3 mutations disrupted root auxin maxima.
- Association Auxin induces ROP3.
- Observation ROP3 is prominently detected in root stele cells.
- Mechanism ROP3 maintains PIN polarity, enabling polar auxin transport and plant patterning.