Claim · model-written summary
The authors suggest PbeNAC72 activates genes related to core signaling pathways and fundamental cellular processes, contributing to positive regulation of Valsa canker resistance.
Not reportedTaxonMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Verbatim
e0“PbeNAC72 activates genes related to core signaling pathways and fundamental cellular processes, which further contribute to the positive regulation of Valsa canker resistance”
Title
Wenhui Wang; Huanhuan Hu; Yatao Li; et al. Horticulturae 2026. doi:10.3390/horticulturae12020171
Other claims from this paper (5)
- Mechanism PbeNAC72, an NAC transcription factor responsive to Valsa canker signals in Pyrus betulifolia, positively regulates resistance in apple and pear fruits.
- Intervention effect Overexpression of PbeNAC72 significantly enhanced resistance to Valsa canker in Duli-G03 (P. betulifolia) suspension cells.
- Intervention effect Overexpression of PbeNAC72 induced reactive oxygen species bursts in Duli-G03 (P. betulifolia) suspension cells.
- Intervention effect Overexpression of PbeNAC72 induced the expression of salicylic acid and jasmonic acid-related genes in Duli-G03 (P. betulifolia) suspension cells.
- Association Weighted gene co-expression network analysis showed PbeNAC72 is primarily linked to genes associated with DNA replication and microtubule-based movement.