Claim · model-written summary
Overexpression of PbeNAC72 induced the expression of salicylic acid and jasmonic acid-related genes in Duli-G03 (P. betulifolia) suspension cells.
- Taxon
- P. betulifolia
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Pyrus betulifoliae0 - Material
- Duli-G03e0
- Treatment
- overexpression of PbeNAC72e0
- Outcome
- expression of salicylic acid and jasmonic acid-related genese0
- Direction
- inducede0
Not reportedGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
._-_Budapest_XI._ker%C3%BClet.JPG?width=500)
Verbatim
e0“In ‘Duli-G03’ (P. betulifolia) suspension cells, overexpression of PbeNAC72 significantly enhanced resistance to Valsa canker and induced reactive oxygen species bursts, as well as the expression of salicylic acid and jasmonic acid-related genes”
Title
Wenhui Wang; Huanhuan Hu; Yatao Li; et al. Horticulturae 2026. doi:10.3390/horticulturae12020171
Other claims from this paper (5)
- Mechanism PbeNAC72, an NAC transcription factor responsive to Valsa canker signals in Pyrus betulifolia, positively regulates resistance in apple and pear fruits.
- Intervention effect Overexpression of PbeNAC72 significantly enhanced resistance to Valsa canker in Duli-G03 (P. betulifolia) suspension cells.
- Intervention effect Overexpression of PbeNAC72 induced reactive oxygen species bursts in Duli-G03 (P. betulifolia) suspension cells.
- Association Weighted gene co-expression network analysis showed PbeNAC72 is primarily linked to genes associated with DNA replication and microtubule-based movement.
- Mechanism The authors suggest PbeNAC72 activates genes related to core signaling pathways and fundamental cellular processes, contributing to positive regulation of Valsa canker resistance.