Claim · model-written summary
cpck2-1 accumulated more salicylic acid.
- Taxon
- Arabidopsis thaliana
Arabidopsis thalianae1 - Material
- cpck2-1 mutante0
- Outcome
- salicylic acid (SA) levelse0
- Direction
- highere0
Not reportedGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“the cpck2-1 mutant accumulated higher salicylic acid (SA) levels”
e1“Arabidopsis thaliana innate immunity”
Title
Lu Rui; Ping Kang; Jing Shao; et al. The Plant Journal 2024. doi:10.1111/tpj.17000
Other claims from this paper (7)
- Mechanism CPCK2 negatively regulates Arabidopsis innate immunity.
- Observation cpck2 mutants had enhanced resistance to powdery mildew.
- Observation cpck2 mutants had enhanced resistance to Pto DC3000.
- Intervention effect Disabling SA biosynthesis or signaling inhibited cpck2-1 disease resistance.
- Mechanism CPCK2 interacted with SABP3, which was required for cpck2-mediated immunity.
- Observation CPCK2 phosphorylated SABP3.
- Mechanism CPCK2-mediated phosphorylation promoted SABP3 S-nitrosylation.