Claim · model-written summary
Δmoset1 lost pathogenicity on wheat.
- Taxon
- wheat host plantse1
- Treatment
- Δmoset1e0
- Interactor
- Δmoset1e0pathogen
- Outcome
- pathogenicity on wheat host plantse0
- Direction
- loste0
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Verbatim
e0“Δmoset1 lost pathogenicity on wheat host plants”
e1“Δmoset1 lost pathogenicity on wheat host plants”
Title
Kieu Thi Minh Pham; Yoshihiro Inoue; Ba Van Vu; et al. PLoS Genetics 2015. doi:10.1371/journal.pgen.1005385
Other claims from this paper (13)
- Observation Eight KMTs contributed to infection processes to varying degrees.
- Intervention effect Moset1 disruption impaired H3K4 methylation and caused the most severe infection morphogenesis defects.
- Intervention effect Exogenous cAMP greatly restored Δmoset1 appressorium formation.
- Intervention effect Exogenous 16-hydroxypalmitic acid greatly restored Δmoset1 appressorium formation.
- Observation Δmoset1 remained infectious on barley cultivar Nigrate.
- Observation Δmoset1 was impaired in vegetative growth.
- Observation Approximately 5% of M. oryzae genes changed H3K4-me2 or -me3 abundance during infection morphogenesis.
- Association H3K4-me2 and -me3 abundance was positively associated with active transcription.
- Observation Lack of MoSET1 up-regulated 1,491 M. oryzae genes in vegetative mycelia.
- Observation Lack of MoSET1 up-regulated 1,385 M. oryzae genes during infection morphogenesis.
- Observation MoSET1 dependence accounted for 1,201 up-regulated genes among mycelium–germination tube DEGs.
- Observation MoSET1 dependence accounted for 882 down-regulated genes among mycelium–germination tube DEGs.
- Observation MoSET1-dependent DEGs were enriched in signal transduction, transport, RNA processing and translation.