Claim · model-written summary
High AroG accumulation caused a dramatic metabolic shift with normal or deleterious morphology.
- Taxon
- tobacco
Nicotiana tabacume1 - Gene
- AroGe0
- Outcome
- deleterious morphological changese0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“the second had high accumulation of the AroG protein with normal, or deleterious morphological changes having a dramatic shift in plant metabolism”
e1“tobacco ( Nicotiana tabacum ) plants were transformed with a bacterial gene (AroG)”
Title
Moran Oliva; Aviv Guy; Gad Galili; et al. Frontiers in Plant Science 2021. doi:10.3389/fpls.2020.604349
Other claims from this paper (11)
- Observation Low AroG expression produced minor metabolic changes and a normal phenotype.
- Intervention effect Transgenic tobacco leaf phenylalanine increased up to 43-fold versus controls.
- Intervention effect Transgenic tobacco leaf tyrosine increased up to 24-fold versus controls.
- Intervention effect Transgenic tobacco leaf tryptophan increased up to 10-fold versus controls.
- Association Phenylpropanoid metabolites were higher alongside increased phenylalanine.
- Intervention effect AroG tobacco showed improved salt tolerance.
- Intervention effect AroG tobacco did not show improved oxidative stress tolerance.
- Intervention effect AroG tobacco did not show improved drought stress tolerance.
- Intervention effect AroG tobacco strongly inhibited parasite development versus heavily infected controls.
- Observation Only a few parasite stems emerged above soil with AroG tobacco.
- Mechanism The authors suggest phenylpropanoid accumulation may delay parasite development.