Claim · model-written summary
3-phenylpropionic acid concentration increased after infection.
- Taxon
- Turtlegrasst0
- Treatment
- Labyrinthula infectione0
- Interactor
- LabyrinthulaLabyrinthulae0pathogent0
- Outcome
- 3-phenylpropionic acide0
- Direction
- increasee0
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Verbatim
e0“succinic acid, 3-phenylpropionic acid, and methyl p-coumarate concentrations were found to significantly increase following Labyrinthula infection”
t0“Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.”
Title
Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427
Other claims from this paper (13)
- Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
- Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
- Observation Host oxygen consumption peaked 48 hours after infection.
- Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
- Observation Caspase-3 activity peaked 48 hours after infection.
- Observation Reactive oxygen species were detected in turtlegrass lesions.
- Observation Nitric oxide was detected in turtlegrass lesions.
- Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
- Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
- Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
- Intervention effect Succinic acid concentration increased after infection.
- Intervention effect Methyl p-coumarate concentration increased after infection.
- Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.