Claim · model-written summary
Ascorbic acid promoted stomatal opening.
- Taxon
- Vicia faba
Vicia fabat0 - Treatment
- ascorbic acide0
- Outcome
- stomatal openinge0
- Direction
- promotede0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“and promoted stomatal opening, as did ascorbic acid (ASA, an important reducing substrate for H2O2 removal).”
t0“Vicia faba”
Title
Xi-Gui Song; Xiao-Ping She; Jun-Min He; et al. Functional Plant Biology 2006. doi:10.1071/fp05232
Other claims from this paper (12)
- Intervention effect Cytokinins reduced guard cell H2O2 in darkness.
- Intervention effect Auxins reduced guard cell H2O2 in darkness.
- Intervention effect Cytokinins induced stomatal opening in darkness.
- Intervention effect Auxins induced stomatal opening in darkness.
- Intervention effect Cytokinins reduced light-associated exogenous H2O2 in guard cells.
- Intervention effect Cytokinins abolished H2O2 generated during a dark period.
- Intervention effect Cytokinins promoted stomatal opening.
- Intervention effect Auxins did not reduce exogenous H2O2.
- Intervention effect Auxins did not abolish H2O2 generated in darkness.
- Intervention effect Auxins did not promote reopening of dark-closed stomata.
- Mechanism Cytokinins probably lower guard cell H2O2 by scavenging.
- Mechanism Auxins limit guard cell H2O2 by restraining its generation.