Claim · model-written summary
The authors suggest PR induction may arise from A. pullulans pathogenesis.
- Taxon
- apple
Malus domesticae1 - Interactor
- A. pullulans
Aureobasidium pullulanse0biocontrol_agent - Outcome
- PR gene inductione0
- Qualifier
- pathogenesis by A. pullulanse0
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.


Verbatim
e0“PR gene induction in flowers may be a result of pathogenesis by A. pullulans”
e1“apple flowers”
Title
Quan Zeng; Kenneth B. Johnson; Salma Mukhtar; et al. Phytopathology 2023. doi:10.1094/phyto-12-22-0452-r
Other claims from this paper (9)
- Intervention effect Blossom Protect induced systemic acquired resistance PR genes in apple flower hypanthia.
- Intervention effect Blossom Protect did not induce induced systemic resistance pathway genes.
- Association PR gene induction coincided with increased plant-derived salicylic acid.
- Intervention effect E. amylovora inoculation suppressed PR expression in untreated flowers.
- Intervention effect Blossom Protect pretreatment offset pathogen immune repression of PR expression.
- Intervention effect Blossom Protect pretreatment prevented infection after pathogen inoculation.
- Observation PR gene induction occurred two days after Blossom Protect treatment.
- Mechanism PR gene induction required direct flower–yeast contact.
- Observation Some treated flowers had deterioration of the hypanthium epidermis.