Claim · model-written summary
RNAi of AtTZF1-3 caused slightly stress-sensitive phenotypes in Arabidopsis.
- Taxon
- Arabidopsis
Arabidopsist0 - Treatment
- RNAi of AtTZF1-3e2
- Gene
- AtTZF1-3e2
- Outcome
- slightly stress-sensitive phenotypese2
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e2“RNAi of AtTZF1-3 caused early germination and slightly stress-sensitive phenotypes”
t0“Arabidopsis”
Title
Pei‐Chi Lin; Marcelo C. Pomeranz; Yusuke Jikumaru; et al. The Plant Journal 2010. doi:10.1111/j.1365-313x.2010.04419.x
Other claims from this paper (12)
- Association AtTZF1 mRNA expression is repressed by a hexokinase-dependent sugar signaling pathway in Arabidopsis.
- Mechanism AtTZF1 acts as a positive regulator of ABA/sugar responses.
- Mechanism AtTZF1 acts as a negative regulator of GA responses.
- Intervention effect RNAi of AtTZF1-3 caused early germination in Arabidopsis.
- Intervention effect Plants over-expressing AtTZF1 were compact.
- Intervention effect Plants over-expressing AtTZF1 flowered late.
- Intervention effect Plants over-expressing AtTZF1 were stress-tolerant.
- Intervention effect The developmental phenotypes of plants over-expressing AtTZF1 were only partially rescued by exogenous GA application.
- Association The enhanced cold and drought tolerance of AtTZF1 over-expressing plants was not associated with increased ABA accumulation.
- Intervention effect Microarray analysis showed that over-expression of AtTZF1 mimics the effects of ABA or GA deficiency on gene expression.
- Intervention effect A gene network centered on the putative peptide hormone encoded by GASA6 was severely repressed by AtTZF1 over-expression.
- Mechanism AtTZF1 may serve as a regulator connecting sugar, ABA, GA and peptide hormone responses.