Claim · model-written summary
Inhibiting ACC synthase suppressed the expansion defect in fei1 fei2 roots.
- Taxon
- Arabidopsis
Arabidopsist0 - Material
- fei1 fei2e0
- Organ / stage
- rootse0
- Treatment
- inhibition of ACC synthasee0
- Gene
- ACC synthasee0
- Outcome
- expansion defecte0
- Direction
- suppressede0
Not reportedCompoundInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“The expansion defect in fei1 fei2 roots was suppressed by inhibition of ACC synthase”
t0“Arabidopsis”
Title
Xu, Shouling Carolina Digital Repository (University of North Carolina at Chapel Hill) 2019. doi:10.17615/9335-9d02
Other claims from this paper (12)
- Mechanism FEI1 and FEI2 act as positive regulators of Arabidopsis cell wall biosynthesis.
- Intervention effect FEI1 and FEI2 mutations disrupt anisotropic expansion.
- Intervention effect FEI1 and FEI2 mutations disrupt cell wall polymer synthesis.
- Association FEI mutations act additively with cellulose biosynthesis inhibitors or disrupting mutations.
- Observation FEI1 is an active protein kinase.
- Intervention effect Kinase-inactive FEI1 fully complemented the fei1 fei2 mutant.
- Intervention effect Disrupting ethylene response did not suppress the fei1 fei2 root expansion defect.
- Association FEI proteins interact directly with ACC synthase.
- Observation A suppressor screen identified nine suppressors representing eight genes or complementation groups.
- Observation shou1 mapped to chromosome 5 and encodes a pentatricopeptide repeat protein.
- Observation Two shou2 alleles mapped to a 47kb region on chromosome 1.
- Mechanism shou2 inhibits root hair elongation.