Claim · model-written summary
The presence of endophytes in F. arundinacea reduced the CO2 effect on N acquisition in P. lanceolata.
- Taxon
- P. lanceolata
Plantago lanceolatae4 - Treatment
- endophytese4
- Interactor
- endophytese4symbiont
- Outcome
- CO 2 effect on N acquisitione4
- Direction
- reducede4
Not reportedMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Using Festuca arundinacea Schreb and Plantago lanceolata L. as model plants”
e4“The presence of endophytes in F. arundinacea , however, reduced the CO 2 effect on N acquisition in P. lanceolata , although it did not affect growth responses of their host plants to elevated CO 2”
Title
XIN CHEN; CONG TU; MICHAEL G. BURTON; et al. Global Change Biology 2007. doi:10.1111/j.1365-2486.2007.01347.x
Other claims from this paper (10)
- Intervention effect Elevated CO2 increased the shoot biomass ratio of P. lanceolata to F. arundinacea in all mixtures.
- Intervention effect Elevated CO2 stimulated the growth of P. lanceolata more than that of F. arundinacea.
- Intervention effect Elevated CO2 increased mycorrhizal root colonization of P. lanceolata.
- Intervention effect Elevated CO2 had no effect on mycorrhizal root colonization of F. arundinacea.
- Intervention effect Mycorrhizae increased the shoot biomass ratio of P. lanceolata to F. arundinacea under elevated CO2.
- Intervention effect Elevated CO2 enhanced 15N and total N uptake of P. lanceolata in the absence of endophytes.
- Intervention effect Mycorrhizae enhanced 15N and total N uptake of P. lanceolata in the absence of endophytes.
- Intervention effect Elevated CO2 had no or negative effects on N acquisition of F. arundinacea.
- Intervention effect Mycorrhizae had no or negative effects on N acquisition of F. arundinacea.
- Intervention effect Endophytes in F. arundinacea did not affect growth responses of their host plants to elevated CO2.