Claim · model-written summary
Overexpression of SbASR4 in Arabidopsis reduced germination rate under salt stress.
- Taxon
- Arabidopsis
Arabidopsise3 - Treatment
- Overexpression of SbASR4e3
- Gene
- SbASR4t0
- Outcome
- germination ratee3
- Direction
- reducede3
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“Functional Characterization of Sorghum SbASR4 in Salt Stress Response”
e3“Overexpression of SbASR4 significantly exacerbated the sensitivity of Arabidopsis to salt stress, as evidenced by reduced germination rate, inhibited root elonga-tion, increased Na⁺ accumulation, and elevated H2O2 levels”
Title
Functional Characterization of Sorghum SbASR4 in Salt Stress Response
Juan Jiang; Haoran Zhang; Yaxuan Sang; et al. CHINESE BULLETIN OF BOTANY 2026. doi:10.3724/cbb-2026-0035
Other claims from this paper (11)
- Intervention effect Overexpression of SbASR4 in Arabidopsis inhibited root elongation under salt stress.
- Intervention effect Overexpression of SbASR4 in Arabidopsis increased Na⁺ accumulation under salt stress.
- Intervention effect Overexpression of SbASR4 in Arabidopsis elevated H2O2 levels under salt stress.
- Intervention effect Silencing SbASR4 in sorghum markedly enhanced plant salt tolerance.
- Intervention effect SbASR4 was significantly downregulated under salt stress and natural water deficit in sorghum.
- Observation SbASR4 localized to the nucleus and plasma membrane in sorghum.
- Observation SbASR4 undergoes rapid degradation under salt stress.
- Intervention effect Silenced SbASR4 lines showed significant upregulation of SOS pathway-related genes, CAT, and SnRK1.1 under salt stress.
- Observation PP2C family gene expression changed in SbASR4-silenced sorghum lines under salt stress.
- Observation SbASR4 interacts with SbSOS2 in sorghum.
- Mechanism SbASR4 negatively regulates plant salt tolerance, potentially through the antioxidant system, ABA signaling pathway and Na⁺ homeostasis.