Claim · model-written summary
Spermine did not increase endogenous salicylic acid.
- Taxon
- Tobacco
Nicotiana tabacumt0 - Treatment
- Spme0
- Outcome
- level of endogenous SAe0
- Direction
- did not increasee0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“Both Tobacco Acidic”
e0“Spm did not increase the level of endogenous SA”
Title
Hiromoto Yamakawa; Hiroshi Kamada; Masaya Satoh; et al. PLANT PHYSIOLOGY 1998. doi:10.1104/pp.118.4.1213
Other claims from this paper (8)
- Observation An acidic pathogenesis-related protein inducer accumulated in infected necrotic tobacco leaf intercellular spaces.
- Observation The induction activity occurred in low molecular mass basic fractions coeluting with authentic spermine.
- Observation Free spermine in necrotic lesion intercellular spaces was 20-fold higher than in healthy leaves.
- Intervention effect Applied spermine induced acidic PR-1 gene expression.
- Intervention effect Spermine treatment increased acidic PR-1, PR-2, PR-3 and PR-5 protein accumulation.
- Intervention effect Salicylic acid treatment did not significantly increase endogenous spermine.
- Intervention effect Spermine treatment reduced the size of virus-induced local lesions.
- Mechanism Authors infer spermine induces acidic defense proteins and virus resistance through a salicylate-independent pathway.