Claim · model-written summary
The MEK2-SIPK cascade activated BR-induced ROS, defense gene expression and viral resistance.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianat0 - Interactor
- Tobacco mosaic virus
Tobacco mosaic virust0pathogen - Gene
- MEK2-SIPKe0
- Outcome
- RBOHB-dependent ROS production, defense gene expression and virus resistancee0
Not reportedMaterialCompoundOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“MEK2-SIPK cascade activated while BES1/BZR1 inhibited RBOHB-dependent ROS production, defense gene expression and virus resistance induced by BRs.”
t0“Tobacco mosaic virus resistance in Nicotiana benthamiana”
Title
Xing-Guang Deng; Tong Zhu; Xing-Ji Peng; et al. Scientific Reports 2016. doi:10.1038/srep20579
Other claims from this paper (9)
- Intervention effect Applied BRs enhanced viral resistance.
- Intervention effect Bikinin increased viral susceptibility.
- Intervention effect Silencing NbBRI1 blocked BR-induced TMV resistance.
- Intervention effect Silencing NbBSK1 blocked BR-induced TMV resistance.
- Intervention effect Silencing NbBES1/BZR1 blocked Bikinin-reduced resistance.
- Intervention effect Silencing NbMEK2 compromised BR-induced viral resistance and defense gene expression.
- Intervention effect Silencing NbSIPK compromised BR-induced viral resistance and defense gene expression.
- Intervention effect Silencing NbRBOHB compromised BR-induced viral resistance and defense gene expression.
- Mechanism BES1/BZR1 inhibited BR-induced ROS, defense gene expression and viral resistance.