Claim · model-written summary
Phosphorylation of a conserved BKI1 tyrosine disrupted the interaction.
- Taxon
- Vitis vinifera
Vitis viniferae1 - Gene
- BKI1se0
- Outcome
- disrupted the interactione0
- Qualifier
- phosphorylation of a conserved tyrosine residuee0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“phosphorylation of a conserved tyrosine residue in BKI1s disrupted the interaction”
e1“host Vitis vinifera BRI1 kinase inhibitor 1 (VvBKI1)”
Title
Xia Lan; Yunxiao Liu; Shiren Song; et al. Molecular Plant Pathology 2019. doi:10.1111/mpp.12790
Other claims from this paper (12)
- Intervention effect PvRXLR131 expression enhanced C. gloeosporioides pathogenicity on grapevine leaves.
- Intervention effect PvRXLR131 expression in Arabidopsis promoted Pseudomonas syringae DC3000 growth.
- Intervention effect PvRXLR131 expression in Arabidopsis promoted P. syringae DC3000 hrcC mutant growth.
- Intervention effect PvRXLR131 expression suppressed defence-related callose deposition in Arabidopsis.
- Intervention effect Transient PvRXLR131 expression suppressed elicitor-triggered cell death in N. benthamiana leaves.
- Intervention effect Transient PvRXLR131 expression inhibited N. benthamiana resistance to P. capsici.
- Observation PvRXLR131 interacted with VvBKI1, NbBKI1 and AtBKI1.
- Observation PvRXLR131 interacted with VvBKI1 at the plasma membrane.
- Mechanism The PvRXLR131 C-terminus was responsible for BKI1 interaction.
- Intervention effect BKI1 silencing eliminated PvRXLR131 virulence function in N. benthamiana.
- Observation PvRXLR131 transgenic plants showed BKI1-overexpression dwarf phenotypes.
- Observation PvRXLR131 transgenic plants suppressed BR and ER signalling.