Claim · model-written summary
NO down-regulated MYC2.
ArabidopsisNot reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“as well as down-regulating MYC2”
e1“NO also influenced JA responses”
e2“Arabidopsis phytoglobin 2 gene”
Title
Mohamed M. Mira; Owen S. D. Wally; Mohamed Elhiti; et al. Journal of Experimental Botany 2016. doi:10.1093/jxb/erw022
Other claims from this paper (14)
- Mechanism AOS and LOX2 expression was activated by NO.
- Mechanism NO elevated JA content in embryogenic tissue.
- Observation Embryo number declined in the aos1-1 mutant.
- Observation Embryo number declined in the pgb2-aos1-1 double mutant.
- Intervention effect Increasing NO did not rescue the reduced embryo number in aos1-1 backgrounds.
- Intervention effect NO up-regulated PDF1 and JAZ1 expression.
- Intervention effect Ectopic JAZ1 expression promoted somatic embryo formation.
- Intervention effect Suppression of MYC2 promoted somatic embryo formation.
- Intervention effect Repression of JAZ1 reduced embryogenic performance.
- Intervention effect Up-regulation of MYC2 reduced embryogenic performance.
- Intervention effect Sustained JAZ1 expression induced transcription of several IAA biosynthetic genes.
- Intervention effect Sustained JAZ1 expression resulted in higher IAA levels in embryogenic cells.
- Mechanism PGB2 suppression increased JA through NO.
- Mechanism Elevated JA repressed MYC2 and induced JAZ1, favoring IAA accumulation and somatic embryos.