Claim · model-written summary
The bp1 signaling change advanced anther-wall PCD.
- Taxon
- rice
Oryza sativat0 - Organ / stage
- anther wallse0
- Outcome
- programmed cell death (PCD) of anther wallse0
- Qualifier
- thereby advancinge0
Not reportedMaterialGeneCompoundInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“thereby advancing programmed cell death (PCD) of anther walls”
t0“rice”
Title
Huiqiong Chen; Shuqing Zhang; Ruiqi Li; et al. The Plant Cell 2023. doi:10.1093/plcell/koad181
Other claims from this paper (9)
- Mechanism BP1 encodes a UGE that regulates UDP-sugar homeostasis and rice anther development.
- Intervention effect Loss of BP1 caused massive UDP-Glc accumulation.
- Intervention effect Loss of BP1 disrupted other UDP-sugars.
- Mechanism Elevated UDP-Glc and its derivatives in bp1 may induce NADPH oxidase gene expression.
- Mechanism Higher UDP-Glc in bp1 may cause premature ROS accumulation.
- Mechanism The bp1 signaling change delayed the end of tapetal degradation.
- Mechanism UDP-Glc accumulation caused abnormal callose degradation and adhesive microspores.
- Mechanism The UDP-sugar metabolism pathway participates in intine formation.
- Mechanism The UDP-sugar metabolism pathway participates in the initial extine framework.