Pseudomonas cerasi 26 claims

species · family Pseudomonadaceae · bacterium · NCBI Taxonomy ↗

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26 claims

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Observationobservedoriginal resultmeasured

Claim · model-written summary

Pseudomonas cerasi caused leaf spotting on four named forest trees.

Taxon: A. tataricumAcer tataricume0
Interactor: Pseudomonas cerasiPseudomonas cerasie0pathogen
Outcome: Leaf spottinge0

Verbatim

e0
“Leaf spotting on A. tataricum, F. pennsylvanica, U. minor and U. laevis was shown to be caused by Pseudomonas cerasi, Pseudomonas congelans, Pseudomonas graminis, Pseudomonas syringae and Pantoea agglomerans both in monoinfection and coinfection”
✓ located verbatim in the source · abstract, characters 898–1143

Title

Bacteria Pseudomonas sp. and Pantoea sp. Are the New Etiological Agents of Diseases on Forest Trees

Elena Porotikova; Natalia Brusnova; Andrei Sushchenko; et al. Plants 2025. doi:10.3390/plants14040563

Other claims from this paper (6)

Observationobservedoriginal resultmeasured

Claim · model-written summary

Intact probable HrpR was annotated in P. cerasi isolates.

Taxon: sweet cherryPrunus aviumt0
Interactor: P. cerasiPseudomonas cerasie0pathogen
Gene: HrpRe0
Outcome: Intact probable regulatory protein HrpRe0

Verbatim

e0
“Intact probable regulatory protein HrpR was annotated in the genomic sequences of all isolates of P. syringae pv. syringae , P. syringae , P. cerasi , and A .”
✓ located verbatim in the source · abstract, characters 657–815

t0
“sweet cherry”
✓ located verbatim in the source · title, characters 104–116

Title

Pathogenicity, phylogenomic, and comparative genomic study of Pseudomonas syringae sensu lato affecting sweet cherry in California

Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. Microbiology Spectrum 2024. doi:10.1128/spectrum.01324-24

Other claims from this paper (18)

Observation Fifty-eight cherry isolates fell within the P. syringae complex and five genomospecies.
Observation Intact probable HrpR was annotated in P. syringae pv. syringae isolates.
Observation Intact probable HrpR was annotated in P. syringae isolates.
Observation Intact probable HrpR was annotated in genomospecies A isolates.
Observation P. viridiflava isolates had atypical probable HrpR.
Observation Syringomycin and syringopeptin genes were found in the other genomospecies but not A or P. viridiflava.
Observation P. syringae pv. syringae caused cankers, leaf spots and fruit lesions in the field.
Observation P. syringae isolates caused only cankers in the field.
Observation P. cerasi isolates caused only cankers.
Observation Some P. viridiflava isolates caused only cankers.
Observation Genomospecies A caused no field symptoms.
Observation All five genomospecies produced dark lesions on detached immature cherry fruit.
Association Genomospecies A lesions were significantly less severe than those of other genomospecies.
Observation P. cerasi was reported as present and pathogenic on California sweet cherry.
Observation P. viridiflava was reported as present and pathogenic on California sweet cherry.
Observation Copper resistance in P. syringae pv. syringae was 47.5%.
Observation No isolates were resistant to kasugamycin.
Recommendation The authors conclude copper cannot effectively control bacterial blast and canker of sweet cherry.

Observationobservedoriginal resultnegativemeasured

Claim · model-written summary

P. cerasi isolates caused only cankers.

Taxon: sweet cherryPrunus aviumt0
Interactor: P. cerasiPseudomonas cerasie0pathogen
Outcome: cankerse0

Verbatim

e0
“In contrast, all isolates of P. syringae and P. cerasi and some isolates of P. viridiflava caused only cankers.”
✓ located verbatim in the source · abstract, characters 1119–1230

t0
“sweet cherry”
✓ located verbatim in the source · title, characters 104–116

Title

Pathogenicity, phylogenomic, and comparative genomic study of Pseudomonas syringae sensu lato affecting sweet cherry in California

Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. Microbiology Spectrum 2024. doi:10.1128/spectrum.01324-24

Other claims from this paper (18)

Observation Fifty-eight cherry isolates fell within the P. syringae complex and five genomospecies.
Observation Intact probable HrpR was annotated in P. syringae pv. syringae isolates.
Observation Intact probable HrpR was annotated in P. syringae isolates.
Observation Intact probable HrpR was annotated in P. cerasi isolates.
Observation Intact probable HrpR was annotated in genomospecies A isolates.
Observation P. viridiflava isolates had atypical probable HrpR.
Observation Syringomycin and syringopeptin genes were found in the other genomospecies but not A or P. viridiflava.
Observation P. syringae pv. syringae caused cankers, leaf spots and fruit lesions in the field.
Observation P. syringae isolates caused only cankers in the field.
Observation Some P. viridiflava isolates caused only cankers.
Observation Genomospecies A caused no field symptoms.
Observation All five genomospecies produced dark lesions on detached immature cherry fruit.
Association Genomospecies A lesions were significantly less severe than those of other genomospecies.
Observation P. cerasi was reported as present and pathogenic on California sweet cherry.
Observation P. viridiflava was reported as present and pathogenic on California sweet cherry.
Observation Copper resistance in P. syringae pv. syringae was 47.5%.
Observation No isolates were resistant to kasugamycin.
Recommendation The authors conclude copper cannot effectively control bacterial blast and canker of sweet cherry.

Observationobservedoriginal resultmeasured

Claim · model-written summary

P. cerasi was reported as present and pathogenic on California sweet cherry.

Taxon: sweet cherryPrunus aviume0
Interactor: Pseudomonas cerasiPseudomonas cerasie0pathogen
Outcome: presence and pathogenicitye0
Setting: Californiae0

Verbatim

e0
“first report of the presence and pathogenicity of genomospecies Pseudomonas cerasi and Pseudomonas viridiflava in California sweet cherry.”
✓ located verbatim in the source · abstract, characters 2170–2308

Title

Pathogenicity, phylogenomic, and comparative genomic study of Pseudomonas syringae sensu lato affecting sweet cherry in California

Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. Microbiology Spectrum 2024. doi:10.1128/spectrum.01324-24

Other claims from this paper (18)

Observation Fifty-eight cherry isolates fell within the P. syringae complex and five genomospecies.
Observation Intact probable HrpR was annotated in P. syringae pv. syringae isolates.
Observation Intact probable HrpR was annotated in P. syringae isolates.
Observation Intact probable HrpR was annotated in P. cerasi isolates.
Observation Intact probable HrpR was annotated in genomospecies A isolates.
Observation P. viridiflava isolates had atypical probable HrpR.
Observation Syringomycin and syringopeptin genes were found in the other genomospecies but not A or P. viridiflava.
Observation P. syringae pv. syringae caused cankers, leaf spots and fruit lesions in the field.
Observation P. syringae isolates caused only cankers in the field.
Observation P. cerasi isolates caused only cankers.
Observation Some P. viridiflava isolates caused only cankers.
Observation Genomospecies A caused no field symptoms.
Observation All five genomospecies produced dark lesions on detached immature cherry fruit.
Association Genomospecies A lesions were significantly less severe than those of other genomospecies.
Observation P. viridiflava was reported as present and pathogenic on California sweet cherry.
Observation Copper resistance in P. syringae pv. syringae was 47.5%.
Observation No isolates were resistant to kasugamycin.
Recommendation The authors conclude copper cannot effectively control bacterial blast and canker of sweet cherry.

Observationobservedoriginal resultmeasured

Claim · model-written summary

P. cerasi was identified as an almond canker pathogen.

Taxon: almondPrunus dulcise0
Interactor: P. cerasiPseudomonas cerasie0pathogen
Outcome: almond canker pathogense0

Verbatim

e0
“We identified Pseudomonas syringae pv. syringae, P. cerasi, and P. viridiflava as almond canker pathogens.”
✓ located verbatim in the source · abstract, characters 441–547

t0
“almond (Prunus dulcis)”
✓ located verbatim in the source · title, characters 87–109

Title

Phylogenomic analyses and comparative genomics of Pseudomonas syringae associated with almond (Prunus dulcis) in California

Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. PLoS ONE 2024. doi:10.1371/journal.pone.0297867

Other claims from this paper (18)

Observation P. syringae pv. syringae was identified as an almond canker pathogen.
Observation P. viridiflava was identified as an almond canker pathogen.
Observation P. syringae pv. syringae caused canker and foliar blast.
Observation P. cerasi caused only cankers.
Observation P. viridiflava caused only cankers and appeared weakly pathogenic.
Observation P. syringae pv. syringae accounted for 75% of pathogenic isolates.
Observation P. cerasi accounted for 8.3% of pathogenic isolates.
Observation P. viridiflava accounted for 16.7% of pathogenic isolates.
Observation Detached leaves developed significant necrosis and browning after P. cerasi infiltration.
Observation Detached leaves developed significant necrosis and browning after P. syringae pv. syringae infiltration.
Observation P. viridiflava had moderate effects in detached leaf bioassays.
Observation P. cerasi genomes lacked key epiphytic fitness genes.
Observation P. viridiflava genomes lacked key epiphytic fitness genes.
Observation P. syringae pv. syringae isolates carried ice nucleation protein associated with the phenotype.
Observation P. cerasi isolates carried ice nucleation protein associated with the phenotype.
Observation Isolates with ctpV resisted copper up to 600 μg/ml.
Observation Isolates without ctpV failed to grow at 200 μg/ml copper.
Observation All isolates were sensitive to kasugamycin at 100μg/ml.

Observationobservedoriginal resultmeasured

Claim · model-written summary

P. cerasi caused only cankers.

Taxon: almondPrunus dulcist0
Interactor: P. cerasiPseudomonas cerasie0pathogen
Outcome: cankerse0
Qualifier: onlye0

Verbatim

e0
“P. cerasi and P. viridiflava only caused cankers”
✓ located verbatim in the source · abstract, characters 634–682

t0
“almond (Prunus dulcis)”
✓ located verbatim in the source · title, characters 87–109

Title

Phylogenomic analyses and comparative genomics of Pseudomonas syringae associated with almond (Prunus dulcis) in California

Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. PLoS ONE 2024. doi:10.1371/journal.pone.0297867

Other claims from this paper (18)

Observation P. syringae pv. syringae was identified as an almond canker pathogen.
Observation P. cerasi was identified as an almond canker pathogen.
Observation P. viridiflava was identified as an almond canker pathogen.
Observation P. syringae pv. syringae caused canker and foliar blast.
Observation P. viridiflava caused only cankers and appeared weakly pathogenic.
Observation P. syringae pv. syringae accounted for 75% of pathogenic isolates.
Observation P. cerasi accounted for 8.3% of pathogenic isolates.
Observation P. viridiflava accounted for 16.7% of pathogenic isolates.
Observation Detached leaves developed significant necrosis and browning after P. cerasi infiltration.
Observation Detached leaves developed significant necrosis and browning after P. syringae pv. syringae infiltration.
Observation P. viridiflava had moderate effects in detached leaf bioassays.
Observation P. cerasi genomes lacked key epiphytic fitness genes.
Observation P. viridiflava genomes lacked key epiphytic fitness genes.
Observation P. syringae pv. syringae isolates carried ice nucleation protein associated with the phenotype.
Observation P. cerasi isolates carried ice nucleation protein associated with the phenotype.
Observation Isolates with ctpV resisted copper up to 600 μg/ml.
Observation Isolates without ctpV failed to grow at 200 μg/ml copper.
Observation All isolates were sensitive to kasugamycin at 100μg/ml.

Observationobservedoriginal resultmeasured

Claim · model-written summary

P. cerasi accounted for 8.3% of pathogenic isolates.

Taxon: almondPrunus dulcist0
Interactor: P. cerasiPseudomonas cerasie0pathogen
Outcome: pathogenic isolatese0
Value: 8.3e0

Not reportedDirection

Verbatim

e0
“P. cerasi and P. viridiflava isolates composed 8.3 and 16.7% of the pathogenic isolates, respectively.”
✓ located verbatim in the source · abstract, characters 908–1010

t0
“almond (Prunus dulcis)”
✓ located verbatim in the source · title, characters 87–109

Title

Phylogenomic analyses and comparative genomics of Pseudomonas syringae associated with almond (Prunus dulcis) in California

Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. PLoS ONE 2024. doi:10.1371/journal.pone.0297867

Other claims from this paper (18)

Observation P. syringae pv. syringae was identified as an almond canker pathogen.
Observation P. cerasi was identified as an almond canker pathogen.
Observation P. viridiflava was identified as an almond canker pathogen.
Observation P. syringae pv. syringae caused canker and foliar blast.
Observation P. cerasi caused only cankers.
Observation P. viridiflava caused only cankers and appeared weakly pathogenic.
Observation P. syringae pv. syringae accounted for 75% of pathogenic isolates.
Observation P. viridiflava accounted for 16.7% of pathogenic isolates.
Observation Detached leaves developed significant necrosis and browning after P. cerasi infiltration.
Observation Detached leaves developed significant necrosis and browning after P. syringae pv. syringae infiltration.
Observation P. viridiflava had moderate effects in detached leaf bioassays.
Observation P. cerasi genomes lacked key epiphytic fitness genes.
Observation P. viridiflava genomes lacked key epiphytic fitness genes.
Observation P. syringae pv. syringae isolates carried ice nucleation protein associated with the phenotype.
Observation P. cerasi isolates carried ice nucleation protein associated with the phenotype.
Observation Isolates with ctpV resisted copper up to 600 μg/ml.
Observation Isolates without ctpV failed to grow at 200 μg/ml copper.
Observation All isolates were sensitive to kasugamycin at 100μg/ml.

Observationobservedoriginal resultsignificant (as stated)measured

Claim · model-written summary

Detached leaves developed significant necrosis and browning after P. cerasi infiltration.

Taxon: almondPrunus dulcist0
Interactor: P. cerasiPseudomonas cerasie0pathogen
Outcome: necrosis and browning of detached leavese0
Comparator: experiments in the fielde0
Setting: Laboratorye0

Verbatim

e0
“Laboratory leaf infiltration bioassays produced results distinct from experiments in the field with both P. cerasi and P. syringae pv. syringae, causing significant necrosis and browning of detached leaves, whereas P. viridiflava conferred moderate effects.”
✓ located verbatim in the source · abstract, characters 1011–1268

t0
“almond (Prunus dulcis)”
✓ located verbatim in the source · title, characters 87–109

Title

Phylogenomic analyses and comparative genomics of Pseudomonas syringae associated with almond (Prunus dulcis) in California

Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. PLoS ONE 2024. doi:10.1371/journal.pone.0297867

Other claims from this paper (18)

Observation P. syringae pv. syringae was identified as an almond canker pathogen.
Observation P. cerasi was identified as an almond canker pathogen.
Observation P. viridiflava was identified as an almond canker pathogen.
Observation P. syringae pv. syringae caused canker and foliar blast.
Observation P. cerasi caused only cankers.
Observation P. viridiflava caused only cankers and appeared weakly pathogenic.
Observation P. syringae pv. syringae accounted for 75% of pathogenic isolates.
Observation P. cerasi accounted for 8.3% of pathogenic isolates.
Observation P. viridiflava accounted for 16.7% of pathogenic isolates.
Observation Detached leaves developed significant necrosis and browning after P. syringae pv. syringae infiltration.
Observation P. viridiflava had moderate effects in detached leaf bioassays.
Observation P. cerasi genomes lacked key epiphytic fitness genes.
Observation P. viridiflava genomes lacked key epiphytic fitness genes.
Observation P. syringae pv. syringae isolates carried ice nucleation protein associated with the phenotype.
Observation P. cerasi isolates carried ice nucleation protein associated with the phenotype.
Observation Isolates with ctpV resisted copper up to 600 μg/ml.
Observation Isolates without ctpV failed to grow at 200 μg/ml copper.
Observation All isolates were sensitive to kasugamycin at 100μg/ml.

Observationobservedoriginal resultmeasured

Claim · model-written summary

P. cerasi genomes lacked key epiphytic fitness genes.

Taxon: almondPrunus dulcist0
Interactor: P. cerasiPseudomonas cerasie0pathogen
Outcome: absence of key epiphytic fitness-related genese0

Verbatim

e0
“Genome mining revealed the absence of key epiphytic fitness-related genes in P. cerasi and P. viridiflava genomic sequences”
✓ located verbatim in the source · abstract, characters 1269–1392

t0
“almond (Prunus dulcis)”
✓ located verbatim in the source · title, characters 87–109

Title

Phylogenomic analyses and comparative genomics of Pseudomonas syringae associated with almond (Prunus dulcis) in California

Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. PLoS ONE 2024. doi:10.1371/journal.pone.0297867

Other claims from this paper (18)

Observation P. syringae pv. syringae was identified as an almond canker pathogen.
Observation P. cerasi was identified as an almond canker pathogen.
Observation P. viridiflava was identified as an almond canker pathogen.
Observation P. syringae pv. syringae caused canker and foliar blast.
Observation P. cerasi caused only cankers.
Observation P. viridiflava caused only cankers and appeared weakly pathogenic.
Observation P. syringae pv. syringae accounted for 75% of pathogenic isolates.
Observation P. cerasi accounted for 8.3% of pathogenic isolates.
Observation P. viridiflava accounted for 16.7% of pathogenic isolates.
Observation Detached leaves developed significant necrosis and browning after P. cerasi infiltration.
Observation Detached leaves developed significant necrosis and browning after P. syringae pv. syringae infiltration.
Observation P. viridiflava had moderate effects in detached leaf bioassays.
Observation P. viridiflava genomes lacked key epiphytic fitness genes.
Observation P. syringae pv. syringae isolates carried ice nucleation protein associated with the phenotype.
Observation P. cerasi isolates carried ice nucleation protein associated with the phenotype.
Observation Isolates with ctpV resisted copper up to 600 μg/ml.
Observation Isolates without ctpV failed to grow at 200 μg/ml copper.
Observation All isolates were sensitive to kasugamycin at 100μg/ml.

Observationobservedoriginal resultmeasured

Claim · model-written summary

P. cerasi isolates carried ice nucleation protein associated with the phenotype.

Taxon: almondPrunus dulcist0
Interactor: P. cerasiPseudomonas cerasie0pathogen
Gene: ice nucleation proteine0
Outcome: ice nucleation phenotypee0

Verbatim

e0
“P. syringae pv. syringae and P. cerasi isolates harboured the ice nucleation protein, which correlated with the ice nucleation phenotype.”
✓ located verbatim in the source · abstract, characters 1469–1606

t0
“almond (Prunus dulcis)”
✓ located verbatim in the source · title, characters 87–109

Title

Phylogenomic analyses and comparative genomics of Pseudomonas syringae associated with almond (Prunus dulcis) in California

Tawanda E. Maguvu; Rosa J. Frias; Alejandro I. Hernandez-Rosas; et al. PLoS ONE 2024. doi:10.1371/journal.pone.0297867

Other claims from this paper (18)

Observation P. syringae pv. syringae was identified as an almond canker pathogen.
Observation P. cerasi was identified as an almond canker pathogen.
Observation P. viridiflava was identified as an almond canker pathogen.
Observation P. syringae pv. syringae caused canker and foliar blast.
Observation P. cerasi caused only cankers.
Observation P. viridiflava caused only cankers and appeared weakly pathogenic.
Observation P. syringae pv. syringae accounted for 75% of pathogenic isolates.
Observation P. cerasi accounted for 8.3% of pathogenic isolates.
Observation P. viridiflava accounted for 16.7% of pathogenic isolates.
Observation Detached leaves developed significant necrosis and browning after P. cerasi infiltration.
Observation Detached leaves developed significant necrosis and browning after P. syringae pv. syringae infiltration.
Observation P. viridiflava had moderate effects in detached leaf bioassays.
Observation P. cerasi genomes lacked key epiphytic fitness genes.
Observation P. viridiflava genomes lacked key epiphytic fitness genes.
Observation P. syringae pv. syringae isolates carried ice nucleation protein associated with the phenotype.
Observation Isolates with ctpV resisted copper up to 600 μg/ml.
Observation Isolates without ctpV failed to grow at 200 μg/ml copper.
Observation All isolates were sensitive to kasugamycin at 100μg/ml.