Claim · model-written summary
CcDof genes are differentially regulated by infection with Phytophthora parasitica.
- Taxon
- Citrus clementina
Citrus ×clementinat0 - Treatment
- infection by Phytophthora parasiticae0
- Interactor
- Phytophthora parasitica
Phytophthora nicotianaee0pathogen - Outcome
- differentially regulatede0
- Direction
- differentially regulatede0
Not reportedComparator
Verbatim
e0“differentially regulated by various phytohormones (including ABA, SA, GA, and MeJA), low temperature stress, and infection by Phytophthora parasitica”
t0“Citrus clementina”
Title
Lina Hu; Yuanda Lv; Yutong Xie; et al. Horticulturae 2026. doi:10.3390/horticulturae12030348
Other claims from this paper (12)
- Observation Twenty-four Dof genes (CcDof1-CcDof24) were identified from the Citrus clementina genome.
- Observation Phylogenetic analysis classified the Citrus clementina Dof proteins into six distinct clades.
- Observation The Citrus clementina Dof proteins showed evolutionary conservation with Dof members from Arabidopsis and tomato.
- Observation Most CcDof genes are intronless or contain only a few introns.
- Observation Promoter analysis revealed cis-regulatory elements associated with light responsiveness, hormone signaling and abiotic/biotic stress responses.
- Observation CcDof genes exhibit tissue-specific expression patterns.
- Intervention effect CcDof genes are differentially regulated by various phytohormones (ABA, SA, GA, MeJA).
- Intervention effect CcDof genes are differentially regulated by low temperature stress.
- Intervention effect Transient overexpression of CcDof4 in citrus leaves significantly enhanced resistance to P. parasitica.
- Intervention effect Transient overexpression of CcDof6 in citrus leaves significantly enhanced resistance to P. parasitica.
- Intervention effect Overexpression of CcDof4 and CcDof6 in citrus leaves was accompanied by upregulation of the SA pathway markers NPR1 and PR1.
- Mechanism The authors interpret CcDof4 and CcDof6 as mediating citrus disease resistance, likely through modulation of the SA signaling pathway.
