Claim · model-written summary
Cs9g12620 was required for optimal development of canker symptoms.
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Verbatim
e0“Transient overexpression and gene silencing revealed that Cs9g12620 was required for the optimal development of canker symptoms.”
t0“citrus”
Title
Xinyu Chen; Huasong Zou; Tao Zhuo; et al. eLife 2024. doi:10.7554/elife.91684
Other claims from this paper (7)
- Intervention effect Xcc infection induced Cs9g12620 transcription in a PthA4-dependent manner.
- Observation PthA4 bound to a putative effector-binding element in the Cs9g12620 promoter.
- Mechanism PthA4 suppressed Cs9g12620 promoter activity.
- Mechanism CsLOB1 bound to the Cs9g12620 promoter and activated expression.
- Intervention effect CsLOB1 silencing significantly reduced Cs9g12620 expression.
- Observation PhtA4 interacted with CsLOB1.
- Mechanism PhtA4 feedback suppressed CsLOB1 induction of Cs9g12620 expression.