Claim · model-written summary
ZmNAC89 may regulate maize saline–alkali tolerance through REDOX and ABA signaling pathways.
- Gene
- ZmNAC89e0
- Outcome
- saline–alkali tolerancee0
- Qualifier
- REDOX pathway and ABA signal transduction pathwaye0
Zea maysNot reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“ZmNAC89 may regulate maize saline–alkali tolerance through the REDOX pathway and ABA signal transduction pathway”
Title
The Transcription Factor ZmNAC89 Gene Is Involved in Salt Tolerance in Maize (Zea mays L.)
Yingying Hu; Chunxiang Li; Runyu Zhou; et al. International Journal of Molecular Sciences 2023. doi:10.3390/ijms242015099
Other claims from this paper (12)
- Observation ZmNAC89 was isolated and characterized as a maize saline–alkaline tolerance gene.
- Observation ZmNAC89 localized in the nucleus.
- Observation ZmNAC89 showed transcriptional activation activity in vitro.
- Intervention effect Saline–alkaline treatment strongly upregulated ZmNAC89 expression.
- Intervention effect Drought treatment strongly upregulated ZmNAC89 expression.
- Intervention effect ABA treatment strongly upregulated ZmNAC89 expression.
- Intervention effect ZmNAC89 overexpression enhanced salt tolerance in transgenic Arabidopsis seedlings.
- Intervention effect ZmNAC89 overexpression enhanced salt tolerance in transgenic maize seedlings.
- Observation GO analysis implicated oxidation–reduction process genes in ZmNAC89-mediated saline–alkaline stress.
- Observation Twenty haplotypes were found in the ZmNAC89 coding region among 140 inbred maize lines.
- Observation Sixteen SNPs were found in the ZmNAC89 coding region among 140 inbred maize lines.
- Observation HAP20 was identified as an excellent ZmNAC89 haplotype.