Claim · model-written summary
Pip alleviated TuMV susceptibility even in NahG plants lacking SA accumulation.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianat0 - Material
- NahG plantse0
- Treatment
- Pip treatmente0
- Interactor
- TuMV
Turnip mosaic viruse0pathogen - Outcome
- susceptibility to TuMVe0
- Direction
- alleviatede0
Not reportedGeneCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“on NahG plants (in which SA cannot accumulate), Pip treatment still alleviated susceptibility to TuMV”
t0“Nicotiana benthamiana”
Title
Shu Wang; Kelei Han; Jiejun Peng; et al. Molecular Plant Pathology 2019. doi:10.1111/mpp.12808
Other claims from this paper (14)
- Observation Virus infection upregulated NbALD1.
- Intervention effect NbALD1 silencing facilitated TuMV infection.
- Intervention effect NbALD1 overexpression enhanced TuMV resistance.
- Intervention effect Applied pipecolic acid enhanced TuMV resistance.
- Intervention effect NbALD1 silencing lowered SA content.
- Intervention effect NbALD1 overexpression raised SA content.
- Intervention effect Pip treatment raised SA content.
- Mechanism SA mediated TuMV resistance and was required for NbALD1-mediated resistance.
- Observation ACC accumulated in NbALD1-silenced plants.
- Intervention effect ACC was reduced by NbALD1 overexpression.
- Intervention effect Pip treatment reduced ACC.
- Intervention effect ACS1 silencing alleviated susceptibility of NbALD1-silenced plants to TuMV.
- Intervention effect ACC application compromised resistance of Pip-treated or NbALD1 transgenic plants.
- Mechanism Authors infer NbALD1 promotes SA defense and suppresses the susceptible ethylene pathway.