Claim · model-written summary
SA mediated TuMV resistance and was required for NbALD1-mediated resistance.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianat0 - Interactor
- TuMV
Turnip mosaic viruse0pathogen - Gene
- NbALD1e0
- Compound
- SAe0
- Outcome
- resistance to TuMVe0
Not reportedMaterialOrgan / stageSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“SA mediated resistance to TuMV and was required for NbALD1-mediated resistance”
t0“Nicotiana benthamiana”
Title
Shu Wang; Kelei Han; Jiejun Peng; et al. Molecular Plant Pathology 2019. doi:10.1111/mpp.12808
Other claims from this paper (14)
- Observation Virus infection upregulated NbALD1.
- Intervention effect NbALD1 silencing facilitated TuMV infection.
- Intervention effect NbALD1 overexpression enhanced TuMV resistance.
- Intervention effect Applied pipecolic acid enhanced TuMV resistance.
- Intervention effect NbALD1 silencing lowered SA content.
- Intervention effect NbALD1 overexpression raised SA content.
- Intervention effect Pip treatment raised SA content.
- Intervention effect Pip alleviated TuMV susceptibility even in NahG plants lacking SA accumulation.
- Observation ACC accumulated in NbALD1-silenced plants.
- Intervention effect ACC was reduced by NbALD1 overexpression.
- Intervention effect Pip treatment reduced ACC.
- Intervention effect ACS1 silencing alleviated susceptibility of NbALD1-silenced plants to TuMV.
- Intervention effect ACC application compromised resistance of Pip-treated or NbALD1 transgenic plants.
- Mechanism Authors infer NbALD1 promotes SA defense and suppresses the susceptible ethylene pathway.