Claim · model-written summary
Secondary TMV infection does not restrict lesion size and number in PAL-suppressed tobacco, unlike control plants undergoing SAR.
- Taxon
- tobacco
Nicotiana tabacume0
- Treatment
- secondary infection with TMVe1
- Outcome
- restriction of lesion size and numbere1
- Direction
- no_changeinferrede1
- Comparator
- control plantse1
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“tobacco ( Nicotiana tabacum L. cv. Xanthinc) plants”
e1“secondary infection with TMV does not result in the restriction of lesion size and number seen in control plants undergoing systemic acquired resistance (SAR)”
Title
Jacqueline A. Pallas; Nancy L. Paiva; Chris Lamb; et al. The Plant Journal 1996. doi:10.1046/j.1365-313x.1996.10020281.x
Other claims from this paper (12)
- Intervention effect Lesions on PAL-suppressed tobacco develop at the same rate as on control tobacco after TMV infection.
- Observation Lesions induced by TMV on PAL-suppressed plants differ markedly in appearance from those on control plants.
- Observation PAL-suppressed tobacco lesions show a reduced deposition of phenolics.
- Observation PR proteins are induced normally in PAL-suppressed tobacco upon primary infection.
- Observation Free salicylic acid levels in primary inoculated leaves of PAL-suppressed tobacco are approximately fourfold lower than in control plants after 84 h.
- Observation PR proteins are not induced in systemic leaves of PAL-suppressed tobacco.
- Observation The SAR response can be transmitted from a PAL-suppressed rootstock.
- Observation SAR is not observed when the scion is PAL-suppressed.
- Observation Treatment of PAL-suppressed tobacco with dichloro-isonicotinic acid induces PR protein expression and SAR against TMV.
- Observation Treatment with SA only partially restores SAR in PAL-suppressed tobacco.
- Mechanism De novo synthesis of SA and/or other phenylpropanoids is required for expression of resistance in systemic leaves.
- Mechanism The amount of pre-existing phenylpropanoid compounds in systemic leaves may be important for the establishment of SAR.