Claim · model-written summary
Abscisic acid induced BSN.
VitisNot reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“grapevine”
e0“Other inducers were ammonium sulfate, abscisic acid (ABA)”
e1“The most potent inducer of BSN was the amine agmatine”
Title
B.P. HOLZAPFEL; B.G. COOMBE Australian Journal of Grape and Wine Research 1998. doi:10.1111/j.1755-0238.1998.tb00135.x
Other claims from this paper (13)
- Intervention effect Chemical wick feeding induced bunchstem necrosis in grape bunches.
- Intervention effect Chemical wick feeding hastened or delayed berry ripening.
- Intervention effect Ammonium ion delayed ripening.
- Intervention effect Agmatine was the most potent inducer of BSN.
- Intervention effect BTOA induced BSN-like symptoms.
- Intervention effect Ammonium sulfate induced BSN.
- Association Released ammonium levels were uncorrelated with BSN incidence at two weeks.
- Intervention effect Agmatine increased bunchstem ammonium.
- Intervention effect Agmatine increased BSN.
- Mechanism Agmatine potency may arise from release of potentially phytotoxic putrescine.
- Association Extracted ABA and BSN incidence were positively correlated.
- Intervention effect Most perfusions inducing BSN increased ABA.
- Intervention effect Calcium and magnesium salt co-perfusion did not significantly reduce induced BSN incidence.