Claim · model-written summary
NO homeostasis mutants had impaired phyllotaxis and inflorescence stem elongation.
- Taxon
- Arabidopsis thaliana
Arabidopsis thalianae1 - Material
- NO homeostasis mutantse0
- Outcome
- phyllotactic patterns, inflorescence stem elongatione0
- Direction
- impairede0
Not reportedGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“NO homeostasis mutants are impaired in several shoot architectural parameters, including phyllotactic patterns, inflorescence stem elongation, silique production, leaf number, and margin.”
e1“Arabidopsis thaliana”
Title
Nitric Oxide Alters the Pattern of Auxin Maxima and PIN-FORMED1 During Shoot Development
Inmaculada Sánchez-Vicente; Tamara Lechón; María Fernández-Marcos; et al. Frontiers in Plant Science 2021. doi:10.3389/fpls.2021.630792
Other claims from this paper (7)
- Observation NO homeostasis mutants had impaired silique production, leaf number and margins.
- Observation Introducing cue1 into axr1 and pin1 backgrounds exacerbated the NO–auxin relationship phenotypes.
- Observation NO-deficient mutant backgrounds increased auxin maxima.
- Intervention effect NO scavenging increased auxin maxima.
- Mechanism The authors identify NOA1 as the main player associated with NO production in auxin-maxima patterning.
- Mechanism NO controlled PIN1 protein accumulation during leaf development.
- Observation NO overproduction had opposite effects on root and shoot architecture in terms of PIN1 accumulation.