Claim · model-written summary
MET1 silencing conferred cold tolerance.
CitrusNot reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingDirectionValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Silencing of MET1 suppressed CpG methylation, but upregulated TET8 and conferred cold tolerance.”
t0“Citrus”
Title
Yilei Wang; Xin Jiang; Yue Wang; et al. New Phytologist 2025. doi:10.1111/nph.70464
Other claims from this paper (13)
- Association Thirty-three genes showed greater upregulation and hypomethylation in Ichang papeda than lemon.
- Association Thirty-three genes showed more pronounced hypomethylation in Ichang papeda than lemon.
- Observation TET8 showed the most substantial expression and methylation changes.
- Observation TET8 was localized in the plasma membrane and endoplasmic reticulum.
- Mechanism TET8 played a positive role in citrus cold tolerance.
- Observation TET8 methylation was mainly CpG in the exon and 3 untranslated region.
- Intervention effect 5-Azacytidine inhibited CpG methylation in TET8.
- Intervention effect 5-Azacytidine significantly upregulated TET8.
- Intervention effect 5-Azacytidine enhanced cold tolerance.
- Association MET1 expression was lower in Ichang papeda than lemon.
- Intervention effect MET1 silencing suppressed CpG methylation.
- Intervention effect MET1 silencing upregulated TET8.
- Mechanism The authors infer that MET1-mediated CpG methylation in TET8 modulates cold tolerance.