Claim · model-written summary
RhWRKY33 silencing significantly delayed senescence across treatments.
- Taxon
- Rose Plantst0
- Treatment
- silencing of RhWRKY33e0
- Gene
- RhWRKY33e0
- Outcome
- senescence processe0
- Direction
- delayede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Verbatim
e0“silencing of RhWRKY33 significantly delayed the senescence process in the different treatments (control, wounding, ethylene, and EW)”
t0“Rose Plants”
Title
Weikun Jing; Qingcui Zhao; Shuai Zhang; et al. Frontiers in Plant Science 2021. doi:10.3389/fpls.2021.726797
Other claims from this paper (7)
- Observation Transcriptome analysis identified 2,442 DEGs between ethylene and control.
- Observation Transcriptome analysis identified 4,055 DEGs between EW and control.
- Observation Network analysis identified RhWRKY33 as a hub gene.
- Observation RhWRKY33 accumulated in the nucleus.
- Intervention effect RhWRKY33 expression was higher in wounding, ethylene and combined treatments than controls.
- Intervention effect The silencing delay under ethylene and combined treatment was stronger than under wounding.
- Mechanism The authors infer RhWRKY33 promotes floral senescence through ethylene and wounding signalling, mainly ethylene.