Claim · model-written summary
Several hormone and inhibitor treatments did not appreciably change radicle emergence.
- Taxon
- sugar beet
Beta vulgaris subsp. vulgarise0 - Treatment
- gibberellins, brassinosteroids, auxins, cytokinins, and jasmonates or corresponding hormone biosynthesis inhibitorse0
- Outcome
- radicle emergencee0
- Direction
- did not appreciably affecte0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Treatment of sugar beet fruits and seeds with gibberellins, brassinosteroids, auxins, cytokinins, and jasmonates or corresponding hormone biosynthesis inhibitors did not appreciably affect radicle emergence of fruits or seeds.”
Title
Katrin Hermann; Juliane Meinhard; Peter Dobrev; et al. Journal of Experimental Botany 2007. doi:10.1093/jxb/erm162
Other claims from this paper (14)
- Intervention effect Ethylene promoted radicle emergence.
- Intervention effect ACC promoted radicle emergence.
- Intervention effect ABA inhibited seed radicle emergence but scarcely affected fruits.
- Observation Endogenous ACC and ABA in dry mature fruits declined early during imbibition.
- Intervention effect ABA treatment induced seed ACC accumulation.
- Intervention effect ACC treatment did not affect seed ABA content.
- Observation ACO and CYP707A transcripts accumulated during imbibition.
- Intervention effect ABA, ACC and pericarp did not affect seed CYP707A transcripts.
- Intervention effect ABA promoted seed ACO transcript accumulation.
- Intervention effect Pericarp removal promoted seed ACO transcript accumulation.
- Intervention effect ACC did not promote seed ACO transcripts.
- Mechanism An embryo-mediated ABA extrusion system keeps seed ABA content low.
- Mechanism The pericarp restricts ACC leaching during imbibition.
- Mechanism Radicle emergence appears controlled by pericarp, hormone leaching and ABA–ethylene antagonism.