Claim · model-written summary
StERF3 silencing activated defense genes PR1, NPR1 and WRKY1.
- Taxon
- potato
Solanum tuberosume0 - Treatment
- silencing of StERF3e0
- Outcome
- activation of defense-related genes (PR1, NPR1 and WRKY1)e0
- Direction
- activatione0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“silencing of StERF3 in potato produced an enhanced foliage resistance to P. infestans and elevated plant tolerance to NaCl stress accompanied by the activation of defense-related genes (PR1, NPR1 and WRKY1)”
Title
Zhendong Tian; Qin He; Haixia Wang; et al. Plant and Cell Physiology 2015. doi:10.1093/pcp/pcv025
Other claims from this paper (12)
- Observation StERF3 has an EAR motif in its C-terminal region.
- Mechanism StERF3 negatively regulates resistance to Phytophthora infestans.
- Mechanism StERF3 negatively regulates potato salt tolerance.
- Observation The StERF3 promoter responds to salicylic acid, ABA, ethylene and NaCl.
- Observation The StERF3 promoter responds to P. infestans.
- Observation StERF3 bound the GCC box of the HIS3 promoter and activated HIS3 transcription in yeast.
- Intervention effect Silencing StERF3 enhanced foliage resistance to P. infestans.
- Intervention effect Silencing StERF3 elevated potato tolerance to NaCl stress.
- Intervention effect StERF3 overexpression reduced expression of defense genes.
- Intervention effect StERF3 overexpression enhanced susceptibility to P. infestans.
- Observation StERF3 localizes to the nucleus.
- Mechanism StERF3 interacts with other proteins in the cytoplasm, potentially causing relocalization.