View: List Claim type Research area Organism Taxon as written Decade Paper Intervention effect observed original result negative measured Claim · model-written summary
Microcystin- LR inhibited embryogenic callus growth and development .
Microcystin-LR e0 growth and development of embryogenic calli e0 Not reported Comparator
Verbatim
e0 “Microcystin- LR inhibited the growth and development of embryogenic calli ”
✓ located verbatim in the source · abstract, characters 241–313 t0 “Phragmites australis
✓ located verbatim in the source · title, characters 111–131 Other claims from this paper (7) Intervention effect observed original result positive measured Claim · model-written summary
Reducing lariRNAs restored miRNA levels and LR development in lariRNA hyper-accumulating plants.
Reducing lariRNAs e6 microRNA (miRNA) levels and LR development e6 lariRNA hyper-accumulating plants e6 Verbatim
e6 “Reducing lariRNAs restored microRNA (miRNA) levels and LR development in lariRNA hyper-accumulating plants ”
✓ located verbatim in the source · abstract, characters 937–1043 e0 “is required for LR development in Arabidopsis (Arabidopsis thaliana ) ”
✓ located verbatim in the source · abstract, characters 185–253 Other claims from this paper (9) Intervention effect observed original result negative measured Claim · model-written summary
Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development , while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics.
overexpression of prePIP1and prePIP2 e0 Not reported Comparator
Verbatim
e0 “Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development , while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. ”
✓ located verbatim in the source · abstract, characters 674–877 t0 “Arabidopsis
✓ located verbatim in the source · title, characters 111–122 Other claims from this paper (10) Mechanism Here, we show that PIP1 and PIP2 suppress lateral root (LR) development and seedling growth through the RLK7-MPK3/6-LEAFY PETIOLE (LEP) signaling cascade, with LEP acting as a key node inhibiting LR formation and seedling growth. Intervention effect Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Association Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Association Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Mechanism Genetic analyses, coimmunoprecipitation, peptide-binding and kinase assays, rapamycin-induced dimerization, and a BIR3 chimera system collectively revealed that PIP1/2 recognition promoted RLK7 interaction with SOMATIC EMBRYOGENESIS RECEPTOR KINASE coreceptors, triggering MPK3/6-dependent phosphorylation events that inhibit LR development. Mechanism Genetic analyses, coimmunoprecipitation, peptide-binding and kinase assays, rapamycin-induced dimerization, and a BIR3 chimera system collectively revealed that PIP1/2 recognition promoted RLK7 interaction with SOMATIC EMBRYOGENESIS RECEPTOR KINASE coreceptors, triggering MPK3/6-dependent phosphorylation events that inhibit LR development. Mechanism The transcription factor LEP acted downstream of the RLK7-MPK6, inhibiting LR development. Mechanism Loss of LEP function alleviated PIP-induced inhibition of seedling growth and LR formation without compromising immune responses. Mechanism Loss of LEP function alleviated PIP-induced inhibition of seedling growth and LR formation without compromising immune responses. Intervention effect The flg22 peptide suppressed LR formation and seedling growth in part through the PIP1/2-RLK7-MPK6-LEP signaling pathway bifurcating from immune responses downstream of MPK3/6. Mechanism author interpretation original result measured Claim · model-written summary
Well-known LR regulators, the miRNAs, mainly function downstream of lariRNAs.
mainly function downstream of lariRNAs e7 Verbatim
e7 “these well-known regulators of LR development mainly function downstream of lariRNAs ”
✓ located verbatim in the source · abstract, characters 1061–1145 e0 “is required for LR development in Arabidopsis (Arabidopsis thaliana ) ”
✓ located verbatim in the source · abstract, characters 185–253 Other claims from this paper (9) Mechanism observed original result negative measured Claim · model-written summary
The transcription factor LEP acted downstream of the RLK7-MPK6, inhibiting LR development .
Verbatim
e0 “The transcription factor LEP acted downstream of the RLK7-MPK6, inhibiting LR development . ”
✓ located verbatim in the source · abstract, characters 1220–1310 t0 “Arabidopsis
✓ located verbatim in the source · title, characters 111–122 Other claims from this paper (10) Mechanism Here, we show that PIP1 and PIP2 suppress lateral root (LR) development and seedling growth through the RLK7-MPK3/6-LEAFY PETIOLE (LEP) signaling cascade, with LEP acting as a key node inhibiting LR formation and seedling growth. Intervention effect Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Intervention effect Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Association Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Association Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Mechanism Genetic analyses, coimmunoprecipitation, peptide-binding and kinase assays, rapamycin-induced dimerization, and a BIR3 chimera system collectively revealed that PIP1/2 recognition promoted RLK7 interaction with SOMATIC EMBRYOGENESIS RECEPTOR KINASE coreceptors, triggering MPK3/6-dependent phosphorylation events that inhibit LR development. Mechanism Genetic analyses, coimmunoprecipitation, peptide-binding and kinase assays, rapamycin-induced dimerization, and a BIR3 chimera system collectively revealed that PIP1/2 recognition promoted RLK7 interaction with SOMATIC EMBRYOGENESIS RECEPTOR KINASE coreceptors, triggering MPK3/6-dependent phosphorylation events that inhibit LR development. Mechanism Loss of LEP function alleviated PIP-induced inhibition of seedling growth and LR formation without compromising immune responses. Mechanism Loss of LEP function alleviated PIP-induced inhibition of seedling growth and LR formation without compromising immune responses. Intervention effect The flg22 peptide suppressed LR formation and seedling growth in part through the PIP1/2-RLK7-MPK6-LEP signaling pathway bifurcating from immune responses downstream of MPK3/6. Intervention effect observed original result negative measured Claim · model-written summary
Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development , while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics.
Exogenous application of PIP1/2 peptides e0 Not reported Comparator
Verbatim
e0 “Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development , while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. ”
✓ located verbatim in the source · abstract, characters 674–877 t0 “Arabidopsis
✓ located verbatim in the source · title, characters 111–122 Other claims from this paper (10) Mechanism Here, we show that PIP1 and PIP2 suppress lateral root (LR) development and seedling growth through the RLK7-MPK3/6-LEAFY PETIOLE (LEP) signaling cascade, with LEP acting as a key node inhibiting LR formation and seedling growth. Intervention effect Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Association Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Association Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Mechanism Genetic analyses, coimmunoprecipitation, peptide-binding and kinase assays, rapamycin-induced dimerization, and a BIR3 chimera system collectively revealed that PIP1/2 recognition promoted RLK7 interaction with SOMATIC EMBRYOGENESIS RECEPTOR KINASE coreceptors, triggering MPK3/6-dependent phosphorylation events that inhibit LR development. Mechanism Genetic analyses, coimmunoprecipitation, peptide-binding and kinase assays, rapamycin-induced dimerization, and a BIR3 chimera system collectively revealed that PIP1/2 recognition promoted RLK7 interaction with SOMATIC EMBRYOGENESIS RECEPTOR KINASE coreceptors, triggering MPK3/6-dependent phosphorylation events that inhibit LR development. Mechanism The transcription factor LEP acted downstream of the RLK7-MPK6, inhibiting LR development. Mechanism Loss of LEP function alleviated PIP-induced inhibition of seedling growth and LR formation without compromising immune responses. Mechanism Loss of LEP function alleviated PIP-induced inhibition of seedling growth and LR formation without compromising immune responses. Intervention effect The flg22 peptide suppressed LR formation and seedling growth in part through the PIP1/2-RLK7-MPK6-LEP signaling pathway bifurcating from immune responses downstream of MPK3/6. Association observed original result positive measured Claim · model-written summary
Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development , while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics.
mutations in prePIP1 or prePIP2 e0 Verbatim
e0 “Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development , while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. ”
✓ located verbatim in the source · abstract, characters 674–877 t0 “Arabidopsis
✓ located verbatim in the source · title, characters 111–122 Other claims from this paper (10) Mechanism Here, we show that PIP1 and PIP2 suppress lateral root (LR) development and seedling growth through the RLK7-MPK3/6-LEAFY PETIOLE (LEP) signaling cascade, with LEP acting as a key node inhibiting LR formation and seedling growth. Intervention effect Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Intervention effect Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Association Exogenous application of PIP1/2 peptides and overexpression of prePIP1and prePIP2 inhibited LR development, while mutations in prePIP1 or prePIP2 enhanced primary root growth and accelerated LR kinetics. Mechanism Genetic analyses, coimmunoprecipitation, peptide-binding and kinase assays, rapamycin-induced dimerization, and a BIR3 chimera system collectively revealed that PIP1/2 recognition promoted RLK7 interaction with SOMATIC EMBRYOGENESIS RECEPTOR KINASE coreceptors, triggering MPK3/6-dependent phosphorylation events that inhibit LR development. Mechanism Genetic analyses, coimmunoprecipitation, peptide-binding and kinase assays, rapamycin-induced dimerization, and a BIR3 chimera system collectively revealed that PIP1/2 recognition promoted RLK7 interaction with SOMATIC EMBRYOGENESIS RECEPTOR KINASE coreceptors, triggering MPK3/6-dependent phosphorylation events that inhibit LR development. Mechanism The transcription factor LEP acted downstream of the RLK7-MPK6, inhibiting LR development. Mechanism Loss of LEP function alleviated PIP-induced inhibition of seedling growth and LR formation without compromising immune responses. Mechanism Loss of LEP function alleviated PIP-induced inhibition of seedling growth and LR formation without compromising immune responses. Intervention effect The flg22 peptide suppressed LR formation and seedling growth in part through the PIP1/2-RLK7-MPK6-LEP signaling pathway bifurcating from immune responses downstream of MPK3/6. Intervention effect observed original result measured Claim · model-written summary
ABA inhibition of lateral root development occurred immediately after emergence of the lateral root primordium, before activation of the LR meristem, and was reversible.
LR primordium (LRP) e2 Not reported Comparator
Verbatim
e0 “checkpoint for lateral root development in Arabidopsis ”
✓ located verbatim in the source · abstract, characters 1614–1668 e1 “In the presence of exogenous ABA, LR development is inhibited ”
✓ located verbatim in the source · abstract, characters 484–545 e2 “This occurs at a specific development al stage, i.e. immediately after the emergence of the LR primordium ( LR P) from the parent root and prior to the activation of the LR meristem, and is reversible ”
✓ located verbatim in the source · abstract, characters 547–744 Other claims from this paper (10) Observation observed original result negative measured Claim · model-written summary
cat2-1 inhibited lateral root development .
Verbatim
e0 “cat2-1 showed inhibition of LR development and PR elongation, exacerbated by Cd. ”
✓ located verbatim in the source · abstract, characters 939–1019 t0 “Arabidopsis
✓ located verbatim in the source · title, characters 43–54 Other claims from this paper (9) Intervention effect observed original result measured Claim · model-written summary
GR24 eliminated the effect of NAA on secondary LR development in d10 mutants
effect of NAA on secondary LR development e2 Verbatim
e1 “Rice d mutants with impaired SL biosynthesis and signalling exhibited increased secondary LR production compared with wild-type (WT) plants ”
✓ located verbatim in the source · abstract, characters 732–871 e2 “GR24 eliminated the effect of NAA on secondary LR development in the d10 mutants, but not in the d3, mutants ”
✓ located verbatim in the source · abstract, characters 1578–1686 Other claims from this paper (13) Lexical search (BM25). All words must match; if nothing does, any word. Organism names also match every spelling of the same organism. No semantic search yet.
Phragmites australist0
Arabidopsis thaliana
Oryza sativa