Labyrinthulaceae 134 claims

family · order Labyrinthulida · 3 taxa · NCBI Taxonomy ↗

3 entries

Labyrinthula zosterae (nom. inval.)also Labyrinthula zosterae · also L. zosterae50 claims · 7 papers Labyrinthula terrestris47 claims · 10 papers Labyrinthula37 claims · 10 papers

134 claims

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Intervention effectobservedoriginal resultnegativemeasured

Claim · model-written summary

Labyrinthula infection induced lesion progression in turtlegrass.

Taxon: Thalassia testudinum Banks ex KönigThalassia testudinume0
Treatment: Infectione0
Interactor: Labyrinthula sp.Labyrinthulae0pathogent0
Outcome: lesion progressione0
Direction: inducee0

Not reportedComparator

Verbatim

e0
“Infection of the marine subtropical seagrass Thalassia testudinum Banks ex König by pathogenic Labyrinthula sp. was found to induce lesion progression”
✓ located verbatim in the source · abstract, characters 0–150

t0
“pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 104–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
Observation Host oxygen consumption peaked 48 hours after infection.
Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
Observation Caspase-3 activity peaked 48 hours after infection.
Observation Reactive oxygen species were detected in turtlegrass lesions.
Observation Nitric oxide was detected in turtlegrass lesions.
Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.

Intervention effectobservedoriginal resultmeasured

Claim · model-written summary

Infection altered host oxidative metabolism and induced defense metabolites.

Taxon: Thalassia testudinumThalassia testudinumt0
Treatment: infectione0
Interactor: Labyrinthula sp.Labyrinthulat0pathogen
Outcome: host's oxidative metabolism, and production of defense metabolitese0

Not reportedComparatorDirection

Verbatim

e0
“alterations to the host's oxidative metabolism, and production of defense metabolites over the early stages of infection”
✓ located verbatim in the source · abstract, characters 152–272

t0
“Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 13–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
Observation Host oxygen consumption peaked 48 hours after infection.
Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
Observation Caspase-3 activity peaked 48 hours after infection.
Observation Reactive oxygen species were detected in turtlegrass lesions.
Observation Nitric oxide was detected in turtlegrass lesions.
Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.

Observationobservedoriginal resultmeasured

Claim · model-written summary

Host oxygen consumption peaked 48 hours after infection.

Taxon: Thalassia testudinumThalassia testudinumt0
Interactor: Labyrinthula sp.Labyrinthulat0pathogen
Outcome: host oxygen consumptione0
Direction: higheste0
Qualifier: 48-hr post-infectione0

Verbatim

e0
“By 48-hr post-infection, host oxygen consumption, internal reactive oxygen concentrations, and caspase-3 proteolytic activity reached their highest levels”
✓ located verbatim in the source · abstract, characters 311–465

t0
“Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 13–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
Observation Caspase-3 activity peaked 48 hours after infection.
Observation Reactive oxygen species were detected in turtlegrass lesions.
Observation Nitric oxide was detected in turtlegrass lesions.
Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.

Observationobservedoriginal resultmeasured

Claim · model-written summary

Internal reactive oxygen concentrations peaked 48 hours after infection.

Taxon: Thalassia testudinumThalassia testudinumt0
Interactor: Labyrinthula sp.Labyrinthulat0pathogen
Outcome: internal reactive oxygen concentrationse0
Direction: higheste0
Qualifier: 48-hr post-infectione0

Verbatim

e0
“By 48-hr post-infection, host oxygen consumption, internal reactive oxygen concentrations, and caspase-3 proteolytic activity reached their highest levels”
✓ located verbatim in the source · abstract, characters 311–465

t0
“Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 13–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
Observation Host oxygen consumption peaked 48 hours after infection.
Observation Caspase-3 activity peaked 48 hours after infection.
Observation Reactive oxygen species were detected in turtlegrass lesions.
Observation Nitric oxide was detected in turtlegrass lesions.
Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.

Observationobservedoriginal resultmeasured

Claim · model-written summary

Caspase-3 activity peaked 48 hours after infection.

Taxon: Thalassia testudinumThalassia testudinumt0
Interactor: Labyrinthula sp.Labyrinthulat0pathogen
Gene: caspase-3e0
Outcome: caspase-3 proteolytic activitye0
Direction: higheste0
Qualifier: 48-hr post-infectione0

Verbatim

e0
“By 48-hr post-infection, host oxygen consumption, internal reactive oxygen concentrations, and caspase-3 proteolytic activity reached their highest levels”
✓ located verbatim in the source · abstract, characters 311–465

t0
“Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 13–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
Observation Host oxygen consumption peaked 48 hours after infection.
Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
Observation Reactive oxygen species were detected in turtlegrass lesions.
Observation Nitric oxide was detected in turtlegrass lesions.
Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.

Observationobservedoriginal resultmeasured

Claim · model-written summary

Reactive oxygen species were detected in turtlegrass lesions.

Taxon: T. testudinumThalassia testudinume0
Organ / stage: lesionse0
Interactor: Labyrinthula sp.Labyrinthulat0pathogen
Outcome: reactive oxygen species (ROS)e0

Not reportedDirection

Verbatim

e0
“The formation of reactive oxygen species (ROS) and nitric oxide (NO) were detected in T. testudinum lesions”
✓ located verbatim in the source · abstract, characters 467–574

t0
“pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 104–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
Observation Host oxygen consumption peaked 48 hours after infection.
Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
Observation Caspase-3 activity peaked 48 hours after infection.
Observation Nitric oxide was detected in turtlegrass lesions.
Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.

Observationobservedoriginal resultmeasured

Claim · model-written summary

Nitric oxide was detected in turtlegrass lesions.

Taxon: T. testudinumThalassia testudinume0
Organ / stage: lesionse0
Interactor: Labyrinthula sp.Labyrinthulat0pathogen
Outcome: nitric oxide (NO)e0

Not reportedDirection

Verbatim

e0
“The formation of reactive oxygen species (ROS) and nitric oxide (NO) were detected in T. testudinum lesions”
✓ located verbatim in the source · abstract, characters 467–574

t0
“pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 104–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
Observation Host oxygen consumption peaked 48 hours after infection.
Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
Observation Caspase-3 activity peaked 48 hours after infection.
Observation Reactive oxygen species were detected in turtlegrass lesions.
Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.

Intervention effectobservedoriginal resultmeasured

Claim · model-written summary

Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.

Taxon: Thalassia testudinumThalassia testudinumt0
Treatment: diphenyleneiodoniume0
Interactor: Labyrinthula sp.Labyrinthulat0pathogen
Outcome: Hydrogen peroxide production and lesion expansione0

Not reportedComparatorDirection

Verbatim

e0
“Hydrogen peroxide production and lesion expansion were highly regulated in the presence of diphenyleneiodonium, catalase, and c-PTIO”
✓ located verbatim in the source · abstract, characters 793–925

t0
“Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 13–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
Observation Host oxygen consumption peaked 48 hours after infection.
Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
Observation Caspase-3 activity peaked 48 hours after infection.
Observation Reactive oxygen species were detected in turtlegrass lesions.
Observation Nitric oxide was detected in turtlegrass lesions.
Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.

Intervention effectobservedoriginal resultmeasured

Claim · model-written summary

Catalase regulated hydrogen peroxide production and lesion expansion.

Taxon: Thalassia testudinumThalassia testudinumt0
Treatment: catalasee0
Interactor: Labyrinthula sp.Labyrinthulat0pathogen
Outcome: Hydrogen peroxide production and lesion expansione0

Not reportedComparatorDirection

Verbatim

e0
“Hydrogen peroxide production and lesion expansion were highly regulated in the presence of diphenyleneiodonium, catalase, and c-PTIO”
✓ located verbatim in the source · abstract, characters 793–925

t0
“Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 13–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
Observation Host oxygen consumption peaked 48 hours after infection.
Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
Observation Caspase-3 activity peaked 48 hours after infection.
Observation Reactive oxygen species were detected in turtlegrass lesions.
Observation Nitric oxide was detected in turtlegrass lesions.
Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
Intervention effect c-PTIO regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.

Intervention effectobservedoriginal resultmeasured

Claim · model-written summary

c-PTIO regulated hydrogen peroxide production and lesion expansion.

Taxon: Thalassia testudinumThalassia testudinumt0
Treatment: c-PTIOe0
Interactor: Labyrinthula sp.Labyrinthulat0pathogen
Outcome: Hydrogen peroxide production and lesion expansione0

Not reportedComparatorDirection

Verbatim

e0
“Hydrogen peroxide production and lesion expansion were highly regulated in the presence of diphenyleneiodonium, catalase, and c-PTIO”
✓ located verbatim in the source · abstract, characters 793–925

t0
“Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.”
✓ located verbatim in the source · title, characters 13–131

Title

Turtlegrass (Thalassia testudinum) undergoes a coordinated hypersensitive response when challenged with pathogenic Labyrinthula sp.

Ava Larson; Daniel L. Martin; Cliff Ross Plant Physiology and Biochemistry 2025. doi:10.1016/j.plaphy.2025.110427

Other claims from this paper (13)

Intervention effect Labyrinthula infection induced lesion progression in turtlegrass.
Intervention effect Infection altered host oxidative metabolism and induced defense metabolites.
Observation Host oxygen consumption peaked 48 hours after infection.
Observation Internal reactive oxygen concentrations peaked 48 hours after infection.
Observation Caspase-3 activity peaked 48 hours after infection.
Observation Reactive oxygen species were detected in turtlegrass lesions.
Observation Nitric oxide was detected in turtlegrass lesions.
Intervention effect Diphenyleneiodonium regulated hydrogen peroxide production and lesion expansion.
Intervention effect Catalase regulated hydrogen peroxide production and lesion expansion.
Intervention effect Succinic acid concentration increased after infection.
Intervention effect 3-phenylpropionic acid concentration increased after infection.
Intervention effect Methyl p-coumarate concentration increased after infection.
Mechanism The authors attribute lesions to host hypersensitive cell death and direct pathogen tissue degradation.