Claim · model-written summary
Genes responsive to MCA may play crucial roles in maintaining cell wall integrity to enhance fruit resistance to CGM.
CitrusNot reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingDirectionValue
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“citrus”
e0“these genes responsive to MCA may play crucial roles in maintaining cell wall integrity to enhance fruit resistance to CGM”
Title
Qiang Huang; Yajie Zhang; Lulu Huang; et al. Journal of Future Foods 2026. doi:10.1016/j.jfutfo.2026.04.008
Other claims from this paper (14)
- Intervention effect 0.4 g/L MCA treatment slowed the progression of P. digitatum infection in harvested navel orange.
- Intervention effect 0.4 g/L MCA treatment reduced the severity of citrus green mold in harvested navel orange.
- Intervention effect MCA decreased the activities of cell wall-degrading enzymes.
- Intervention effect MCA maintained cell wall integrity.
- Intervention effect MCA treatment significantly diminished the number of DEGs in P. digitatum-infected fruits compared with healthy fruits.
- Observation A total of 432 DEGs were identified among the three groups.
- Observation 42 of the DEGs were associated with cell wall degradation.
- Observation 29 transcription factors were related to CGM resistance.
- Intervention effect Expression of CsPME3, CsPG2 and CsCLE1 was up-regulated by MCA treatment.
- Intervention effect Expression of CsXTH9 was up-regulated by MCA treatment.
- Intervention effect Expression of most of the 10 TFs was up-regulated by MCA treatment.
- Observation Expression of CsGal6, CsMYB51 and CsWRKY7 was suppressed during the later stages of P. digitatum infection.
- Association Enhanced resistance to CGM may be linked to ERF017/109, DREB1D, MYB108 and WRKY75.
- Recommendation MCA treatment could be a promising strategy for preventing postharvest fungal diseases in citrus fruits.