Claim · model-written summary
Expression of CsXTH9 was up-regulated by MCA treatment.
CitrusNot reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“citrus”
e0“the expression levels of CsPME3”
e1“CsXTH9 , and most of the 10 TFs were up-regulated by MCA treatment”
Title
Qiang Huang; Yajie Zhang; Lulu Huang; et al. Journal of Future Foods 2026. doi:10.1016/j.jfutfo.2026.04.008
Other claims from this paper (14)
- Intervention effect 0.4 g/L MCA treatment slowed the progression of P. digitatum infection in harvested navel orange.
- Intervention effect 0.4 g/L MCA treatment reduced the severity of citrus green mold in harvested navel orange.
- Intervention effect MCA decreased the activities of cell wall-degrading enzymes.
- Intervention effect MCA maintained cell wall integrity.
- Intervention effect MCA treatment significantly diminished the number of DEGs in P. digitatum-infected fruits compared with healthy fruits.
- Observation A total of 432 DEGs were identified among the three groups.
- Observation 42 of the DEGs were associated with cell wall degradation.
- Observation 29 transcription factors were related to CGM resistance.
- Intervention effect Expression of CsPME3, CsPG2 and CsCLE1 was up-regulated by MCA treatment.
- Intervention effect Expression of most of the 10 TFs was up-regulated by MCA treatment.
- Observation Expression of CsGal6, CsMYB51 and CsWRKY7 was suppressed during the later stages of P. digitatum infection.
- Association Enhanced resistance to CGM may be linked to ERF017/109, DREB1D, MYB108 and WRKY75.
- Mechanism Genes responsive to MCA may play crucial roles in maintaining cell wall integrity to enhance fruit resistance to CGM.
- Recommendation MCA treatment could be a promising strategy for preventing postharvest fungal diseases in citrus fruits.