Claim · model-written summary
NbASN silencing compromised PVX accumulation in N. benthamiana leaves.
- Taxon
- N. benthamiana
Nicotiana benthamianae0 - Treatment
- TRV-induced silencing of NbASNe0
- Interactor
- PVXPotato virus Xe0pathogen
- Gene
- NbASNe0
- Outcome
- PVX accumulatione0
- Direction
- compromisese0
Not reportedMaterialCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“TRV-induced silencing of NbASN, the DIN6 homologue in N. benthamiana, compromises TRV and PVX accumulation in systemically infected leaves”
Title
Lourdes Fernández‐Calvino; Irene Guzmán‐Benito; Francisco J. del Toro; et al. Molecular Plant Pathology 2015. doi:10.1111/mpp.12257
Other claims from this paper (10)
- Observation Arabidopsis TRV infection and senescence had extensive overlapping transcriptional responses.
- Observation AtDIN1, AtDIN6 and AtDIN11 were upregulated during infection.
- Observation DIN homologues were activated in N. benthamiana by TRV.
- Observation DIN homologues were activated in N. benthamiana by PVX.
- Intervention effect AtDIN11 RNAi reduced TRV levels in Arabidopsis.
- Intervention effect TRV accumulation was low in AtDIN11 RNAi Arabidopsis protoplasts.
- Observation DIN6 had negligible effect on virus accumulation in Arabidopsis.
- Intervention effect NbASN silencing compromised TRV accumulation in N. benthamiana leaves.
- Association NbASN inactivation correlated with morphological defects in infected leaves.
- Mechanism DIN6 and DIN11 regulated virus multiplication before plant defence activation.