Claim · model-written summary
Silencing NbSGS3 facilitated PVX pathogenesis.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianae0 - Treatment
- Silencing of NbSGS3e1
- Interactor
- PVXPotato virus Xe1pathogen
- Gene
- NbSGS3e1
- Outcome
- PVX pathogenesise1
- Direction
- facilitatede1
Not reportedMaterialCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Nicotiana benthamiana plants”
e1“Silencing of NbSGS3 facilitated PVX pathogenesis.”
Title
Dibyendu Ghosh; M. Malavika; Pratibha Rawat; et al. Virology Journal 2025. doi:10.1186/s12985-025-03005-0
Other claims from this paper (7)
- Mechanism PepLCBV V2 promotes PVX accumulation.
- Intervention effect Mixed PVX and PepLCBV infection aggravated PVX symptoms and increased titre relative to PVX alone.
- Intervention effect PepLCBV without V2 did not increase PVX symptoms.
- Observation PepLCBV V2 suppresses post-transcriptional gene silencing.
- Observation PepLCBV V2 interacts with NbSGS3.
- Intervention effect A silencing-suppressor V2 mutant failed to boost PVX titre.
- Observation Tomato leaf curl New Delhi virus showed a similar association with PVX in N. benthamiana and tomato.