Claim · model-written summary
Suppressing postcold tAPX regulation increased bacterial growth.
- Taxon
- Arabidopsis
Arabidopsise1 - Treatment
- Suppression of tAPX postcold regulatione0
- Gene
- tAPXe0
- Outcome
- bacterial growth numberse0
- Direction
- increasede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Suppression of tAPX postcold regulation in a conditional-inducible tAPX-RNAi line led to increased bacterial growth numbers”
e1“susceptibility of Arabidopsis to virulent Pseudomonas”
Title
Griebel, Thomas; Schütte, Dominic; Ebert, Alina; et al. Refubium (Universitätsbibliothek der Freien Universität Berlin) 2022. doi:10.17169/refubium-35457
Other claims from this paper (9)
- Mechanism Cold priming caused postcold tAPX accumulation and altered transcriptional response to a second cold stimulus for days.
- Intervention effect A 24-h cold treatment at 4°C decreased Arabidopsis susceptibility to virulent Pseudomonas syringae pv. tomato DC3000.
- Intervention effect Cold treatment did not alter Arabidopsis resistance to avirulent P. syringae pv. tomato avRPM1.
- Intervention effect Cold treatment did not alter Arabidopsis resistance to P. syringae pv. tomato avrRPS4.
- Intervention effect Cold treatment did not alter Arabidopsis resistance to effector deficient hrcC−.
- Observation Cold priming protection against P. syringae pv. tomato was immediate and lasted at least five days.
- Mechanism Cold priming benefit was independent of EDS1.
- Mechanism Cold priming benefit was independent of NHL10, FRK1, ICS1 and PR1 activation.
- Mechanism Cold priming benefit required thylakoid bound and stromal ascorbate peroxidase activity.