Claim · model-written summary
Ectopic LecRK-VI.2 expression primed callose deposition and gene expression after flagellin treatment.
- Taxon
- N. benthamiana
Nicotiana benthamianae0 - Treatment
- Ectopic expression of LecRK-VI.2e0
- Outcome
- callose deposition and gene expressione0
- Direction
- primede0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Ectopic expression of LecRK-VI.2 in N. benthamiana primed PTI-mediated reactive oxygen species production, mitogen-activated protein kinase (MAPK) activity, callose deposition and gene expression upon treatment with the MAMP flagellin”
Title
Pin‐Yao Huang; Yu‐Hung Yeh; An‐Chi Liu; et al. The Plant Journal 2014. doi:10.1111/tpj.12557
Other claims from this paper (7)
- Association LecRK-VI.2 associated with FLS2.
- Observation LecRK-VI.2 remained functional after transfer from Arabidopsis to Nicotiana benthamiana.
- Intervention effect Wild tobacco expressing LecRK-VI.2 was more resistant to virulent bacteria.
- Intervention effect Wild tobacco expressing LecRK-VI.2 did not gain resistance to Botrytis cinerea.
- Intervention effect Ectopic LecRK-VI.2 expression primed reactive oxygen species production and MAPK activity after flagellin treatment.
- Observation The authors identify LecRK-VI.2 as a member of the FLS2 receptor complex.
- Recommendation The authors suggest expressing PRR-complex components across species to engineer bacterial disease resistance.