Claim · model-written summary
Iron ions interfered with Xoc-induced reactive oxygen species production.
- Treatment
- iron ionse0
- Interactor
- Xoce0pathogen
- Outcome
- reactive oxygen species productione0
- Direction
- interferese0
Not reportedTaxonMaterialGeneCompoundOrgan / stageSecond interactorSecond interactor roleComparatorSettingValueQualifier
Verbatim
e0“iron ions promote Xoc infection and interferes with reactive oxygen species production induced by Xoc”
Title
Haimiao Zhang; Baolong Sun; Wei Wu; et al. Plant Communications 2024. doi:10.1016/j.xplc.2024.100859
Other claims from this paper (12)
- Intervention effect OsMYBxoc1 overexpression significantly increased rice resistance.
- Intervention effect OsMYBxoc1 inhibited lines and knockout mutants showed the opposite resistance response.
- Intervention effect Suppressing OsMYBxoc1 dampened the PAMP-induced immune response.
- Mechanism OsMYBxoc1 binds the OsNRAMP5 promoter and represses transcription.
- Mechanism OsNRAMP5 negatively regulates rice resistance to BLS.
- Observation OsNRAMP5 has metal ion transport activity.
- Intervention effect Inhibiting OsMYBxoc1 increased iron ion content in rice.
- Intervention effect Ion-dependent H2O2 scavenging activity increased when OsMYBxoc1 expression was inhibited.
- Intervention effect Ion-dependent H2O2 scavenging activity increased with OsNRAMP5 overexpression.
- Intervention effect Iron ions promoted Xoc infection.
- Mechanism The XopAK effector directly inhibits OsMYBxoc1 transcription.
- Intervention effect Iron complemented the pathogenicity defect of the RS105_ΔXopAK mutant strain.